| pubmed-article:9884384 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9884384 | lifeskim:mentions | umls-concept:C0005680 | lld:lifeskim |
| pubmed-article:9884384 | lifeskim:mentions | umls-concept:C0042402 | lld:lifeskim |
| pubmed-article:9884384 | lifeskim:mentions | umls-concept:C0682075 | lld:lifeskim |
| pubmed-article:9884384 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:9884384 | lifeskim:mentions | umls-concept:C2249967 | lld:lifeskim |
| pubmed-article:9884384 | lifeskim:mentions | umls-concept:C0599946 | lld:lifeskim |
| pubmed-article:9884384 | lifeskim:mentions | umls-concept:C1524003 | lld:lifeskim |
| pubmed-article:9884384 | lifeskim:mentions | umls-concept:C0439596 | lld:lifeskim |
| pubmed-article:9884384 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:9884384 | pubmed:dateCreated | 1999-2-10 | lld:pubmed |
| pubmed-article:9884384 | pubmed:abstractText | Vasodilator reactivity is attenuated in normotensive blacks, and this may contribute to their enhanced susceptibility to hypertension and its complications. However, the mechanisms responsible for this phenomenon are unknown. We therefore studied nitric oxide (NO)-dependent and -independent vasorelaxation in healthy blacks and whites to investigate the nature of racial differences in vasodilator function. | lld:pubmed |
| pubmed-article:9884384 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9884384 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9884384 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:9884384 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9884384 | pubmed:issn | 0009-7322 | lld:pubmed |
| pubmed-article:9884384 | pubmed:author | pubmed-author:CannonR... | lld:pubmed |
| pubmed-article:9884384 | pubmed:author | pubmed-author:PanzaJ AJA | lld:pubmed |
| pubmed-article:9884384 | pubmed:author | pubmed-author:CardilloCC | lld:pubmed |
| pubmed-article:9884384 | pubmed:author | pubmed-author:KilcoyneC MCM | lld:pubmed |
| pubmed-article:9884384 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9884384 | pubmed:volume | 99 | lld:pubmed |
| pubmed-article:9884384 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9884384 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9884384 | pubmed:pagination | 90-5 | lld:pubmed |
| pubmed-article:9884384 | pubmed:dateRevised | 2004-11-17 | lld:pubmed |
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| pubmed-article:9884384 | pubmed:articleTitle | Attenuation of cyclic nucleotide-mediated smooth muscle relaxation in blacks as a cause of racial differences in vasodilator function. | lld:pubmed |
| pubmed-article:9884384 | pubmed:affiliation | Cardiology Branch, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Md 20892-1650, USA. | lld:pubmed |
| pubmed-article:9884384 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9884384 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:9884384 | pubmed:publicationType | Controlled Clinical Trial | lld:pubmed |
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