| pubmed-article:9856361 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9856361 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:9856361 | lifeskim:mentions | umls-concept:C0600533 | lld:lifeskim |
| pubmed-article:9856361 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
| pubmed-article:9856361 | lifeskim:mentions | umls-concept:C0028351 | lld:lifeskim |
| pubmed-article:9856361 | lifeskim:mentions | umls-concept:C1801960 | lld:lifeskim |
| pubmed-article:9856361 | lifeskim:mentions | umls-concept:C0857121 | lld:lifeskim |
| pubmed-article:9856361 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:9856361 | pubmed:dateCreated | 1999-3-5 | lld:pubmed |
| pubmed-article:9856361 | pubmed:abstractText | To determine whether the abnormal vascular responses observed in salt-sensitive hypertension are caused by an impairment in vascular nitric oxide function. | lld:pubmed |
| pubmed-article:9856361 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9856361 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9856361 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9856361 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9856361 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:9856361 | pubmed:issn | 0263-6352 | lld:pubmed |
| pubmed-article:9856361 | pubmed:author | pubmed-author:MurakamiHH | lld:pubmed |
| pubmed-article:9856361 | pubmed:author | pubmed-author:NishidaYY | lld:pubmed |
| pubmed-article:9856361 | pubmed:author | pubmed-author:KosakaHH | lld:pubmed |
| pubmed-article:9856361 | pubmed:author | pubmed-author:SenPP | lld:pubmed |
| pubmed-article:9856361 | pubmed:author | pubmed-author:DingJJ | lld:pubmed |
| pubmed-article:9856361 | pubmed:author | pubmed-author:ChenQ HQH | lld:pubmed |
| pubmed-article:9856361 | pubmed:author | pubmed-author:ZhouM SMS | lld:pubmed |
| pubmed-article:9856361 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9856361 | pubmed:volume | 16 | lld:pubmed |
| pubmed-article:9856361 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9856361 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9856361 | pubmed:pagination | 1611-8 | lld:pubmed |
| pubmed-article:9856361 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:9856361 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9856361 | pubmed:articleTitle | Role of nitric oxide in vascular hyper-responsiveness to norepinephrine in hypertensive Dahl rats. | lld:pubmed |
| pubmed-article:9856361 | pubmed:affiliation | Second Department of Physiology, Kagawa Medical University, Miki, Japan. ynishida@kms.ac.jp | lld:pubmed |
| pubmed-article:9856361 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9856361 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:9856361 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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