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pubmed-article:9855592pubmed:abstractTextPreeclampsia is a complication of pregnancy that causes maternal vasoconstriction and hypertension. The disease may progress to eclampsia, which is thought to be related to cerebral vasospasm. Although there is evidence for more than one circulating factor that causes endothelial cell dysfunction in preeclampsia, little work has focused on the possibility that vascular smooth muscle function might be directly stimulated by a circulating factor. The aim of this study was to determine whether such a factor or factors could be detected by the vessels.lld:pubmed
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pubmed-article:9855592pubmed:authorpubmed-author:RedmanC WCWlld:pubmed
pubmed-article:9855592pubmed:authorpubmed-author:ClarkJ FJFlld:pubmed
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pubmed-article:9855592pubmed:authorpubmed-author:BearchellM...lld:pubmed
pubmed-article:9855592pubmed:authorpubmed-author:PyneG JGJlld:pubmed
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pubmed-article:9855592pubmed:volume179lld:pubmed
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pubmed-article:9855592pubmed:pagination1534-8lld:pubmed
pubmed-article:9855592pubmed:dateRevised2006-11-15lld:pubmed
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pubmed-article:9855592pubmed:year1998lld:pubmed
pubmed-article:9855592pubmed:articleTitleVascular smooth muscle oxygen consumption is reversibly stimulated by sera from women with preeclampsia.lld:pubmed
pubmed-article:9855592pubmed:affiliationMedical Research Council Clinical and Biochemical Magnetic Resonance Unit, Department of Biochemistry, University of Oxford, United Kingdom.lld:pubmed
pubmed-article:9855592pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:9855592pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed