| pubmed-article:9851271 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9851271 | lifeskim:mentions | umls-concept:C0003315 | lld:lifeskim |
| pubmed-article:9851271 | lifeskim:mentions | umls-concept:C0085295 | lld:lifeskim |
| pubmed-article:9851271 | lifeskim:mentions | umls-concept:C0024432 | lld:lifeskim |
| pubmed-article:9851271 | lifeskim:mentions | umls-concept:C1550315 | lld:lifeskim |
| pubmed-article:9851271 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:9851271 | lifeskim:mentions | umls-concept:C1516240 | lld:lifeskim |
| pubmed-article:9851271 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:9851271 | pubmed:dateCreated | 1998-12-30 | lld:pubmed |
| pubmed-article:9851271 | pubmed:abstractText | We have investigated the effects of interleukin (IL)-10, IL-4 + granulocyte/macrophage colony-stimulating factor (GM-CSF) and tumour necrosis factor alpha (TNF-alpha) on the phenotype and antigen-presenting capacity of synovial fluid (SF) macrophages from patients with rheumatoid arthritis. | lld:pubmed |
| pubmed-article:9851271 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9851271 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9851271 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:9851271 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:9851271 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9851271 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9851271 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:9851271 | pubmed:issn | 0263-7103 | lld:pubmed |
| pubmed-article:9851271 | pubmed:author | pubmed-author:ToivanenPP | lld:pubmed |
| pubmed-article:9851271 | pubmed:author | pubmed-author:LassilaOO | lld:pubmed |
| pubmed-article:9851271 | pubmed:author | pubmed-author:MöttönenMM | lld:pubmed |
| pubmed-article:9851271 | pubmed:author | pubmed-author:PunnonenJJ | lld:pubmed |
| pubmed-article:9851271 | pubmed:author | pubmed-author:SaarioRR | lld:pubmed |
| pubmed-article:9851271 | pubmed:author | pubmed-author:IsomäkiPP | lld:pubmed |
| pubmed-article:9851271 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9851271 | pubmed:volume | 37 | lld:pubmed |
| pubmed-article:9851271 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9851271 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9851271 | pubmed:pagination | 1207-14 | lld:pubmed |
| pubmed-article:9851271 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:9851271 | pubmed:meshHeading | pubmed-meshheading:9851271-... | lld:pubmed |
| pubmed-article:9851271 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9851271 | pubmed:articleTitle | Interleukin-10 inhibits the capacity of synovial macrophages to function as antigen-presenting cells. | lld:pubmed |
| pubmed-article:9851271 | pubmed:affiliation | Turku Immunology Centre, Department of Medical Microbiology, Turku University, Finland. | lld:pubmed |
| pubmed-article:9851271 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9851271 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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