pubmed-article:9832480 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:9832480 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
pubmed-article:9832480 | lifeskim:mentions | umls-concept:C0190211 | lld:lifeskim |
pubmed-article:9832480 | lifeskim:mentions | umls-concept:C0002965 | lld:lifeskim |
pubmed-article:9832480 | lifeskim:mentions | umls-concept:C0205082 | lld:lifeskim |
pubmed-article:9832480 | lifeskim:mentions | umls-concept:C1155266 | lld:lifeskim |
pubmed-article:9832480 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
pubmed-article:9832480 | pubmed:issue | 22 | lld:pubmed |
pubmed-article:9832480 | pubmed:dateCreated | 1999-1-20 | lld:pubmed |
pubmed-article:9832480 | pubmed:abstractText | Systemic markers of inflammation have been found in unstable angina. Disruption of culprit coronary stenoses may cause a greater inflammatory response in patients with unstable than those with stable angina. We assessed the time course of C-reactive protein (CRP), serum amyloid A protein (SAA), and interleukin-6 (IL-6) after single-vessel PTCA in 30 patients with stable and 56 patients with unstable angina (protocol A). We also studied 12 patients with stable and 15 with unstable angina after diagnostic coronary angiography (protocol B). | lld:pubmed |
pubmed-article:9832480 | pubmed:language | eng | lld:pubmed |
pubmed-article:9832480 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9832480 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:9832480 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9832480 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:9832480 | pubmed:month | Dec | lld:pubmed |
pubmed-article:9832480 | pubmed:issn | 0009-7322 | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:PepysM BMB | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:LiuzziAA | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:RebuzziA GAG | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:MaseriAA | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:CilibertiAA | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:VitelliAA | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:CreaFF | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:BiasucciL MLM | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:AltamuraSS | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:BuffonAA | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:GallimoreJ... | lld:pubmed |
pubmed-article:9832480 | pubmed:author | pubmed-author:CaligiuriGG | lld:pubmed |
pubmed-article:9832480 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:9832480 | pubmed:day | 1 | lld:pubmed |
pubmed-article:9832480 | pubmed:volume | 98 | lld:pubmed |
pubmed-article:9832480 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:9832480 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:9832480 | pubmed:pagination | 2370-6 | lld:pubmed |
pubmed-article:9832480 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:9832480 | pubmed:year | 1998 | lld:pubmed |
pubmed-article:9832480 | pubmed:articleTitle | Enhanced inflammatory response to coronary angioplasty in patients with severe unstable angina. | lld:pubmed |
pubmed-article:9832480 | pubmed:affiliation | Istituto di Cardiologia, Universitá Cattolica, Roma, Italy. | lld:pubmed |
pubmed-article:9832480 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:9832480 | pubmed:publicationType | Clinical Trial | lld:pubmed |
pubmed-article:9832480 | pubmed:publicationType | Controlled Clinical Trial | lld:pubmed |
pubmed-article:9832480 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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