| pubmed-article:9767545 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9767545 | lifeskim:mentions | umls-concept:C0006684 | lld:lifeskim |
| pubmed-article:9767545 | lifeskim:mentions | umls-concept:C0011881 | lld:lifeskim |
| pubmed-article:9767545 | lifeskim:mentions | umls-concept:C0033687 | lld:lifeskim |
| pubmed-article:9767545 | lifeskim:mentions | umls-concept:C0003015 | lld:lifeskim |
| pubmed-article:9767545 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:9767545 | lifeskim:mentions | umls-concept:C0205195 | lld:lifeskim |
| pubmed-article:9767545 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:9767545 | pubmed:dateCreated | 1998-12-31 | lld:pubmed |
| pubmed-article:9767545 | pubmed:abstractText | The degree of proteinuria in patients with diabetes correlates strongly with both an increase in progression of nephropathy as well as cardiovascular events. Moreover, post hoc analyses of recent clinical trials support the concept that reductions of blood pressure and proteinuria correlate with a slowed progression of nephropathy. Both angiotensin converting enzyme (ACE) inhibitors and the nondihydropyridine calcium antagonists, (non-DHPCAs) reduce both arterial pressure and proteinuria in those with diabetic nephropathy. | lld:pubmed |
| pubmed-article:9767545 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9767545 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9767545 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9767545 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9767545 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:9767545 | pubmed:issn | 0085-2538 | lld:pubmed |
| pubmed-article:9767545 | pubmed:author | pubmed-author:McMahonF GFG | lld:pubmed |
| pubmed-article:9767545 | pubmed:author | pubmed-author:DeQuattroVV | lld:pubmed |
| pubmed-article:9767545 | pubmed:author | pubmed-author:WeilM LML | lld:pubmed |
| pubmed-article:9767545 | pubmed:author | pubmed-author:BakrisG LGL | lld:pubmed |
| pubmed-article:9767545 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9767545 | pubmed:volume | 54 | lld:pubmed |
| pubmed-article:9767545 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9767545 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9767545 | pubmed:pagination | 1283-9 | lld:pubmed |
| pubmed-article:9767545 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:9767545 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9767545 | pubmed:articleTitle | Effects of an ACE inhibitor/calcium antagonist combination on proteinuria in diabetic nephropathy. | lld:pubmed |
| pubmed-article:9767545 | pubmed:affiliation | Rush University Hypertension Center, Rush Presbyterian/St. Luke's Medical Center, Chicago, Illinois 60612, USA. gbakris@rush.edu | lld:pubmed |
| pubmed-article:9767545 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9767545 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:9767545 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:9767545 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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