| pubmed-article:9742979 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9742979 | lifeskim:mentions | umls-concept:C0302592 | lld:lifeskim |
| pubmed-article:9742979 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:9742979 | lifeskim:mentions | umls-concept:C0009221 | lld:lifeskim |
| pubmed-article:9742979 | lifeskim:mentions | umls-concept:C0079419 | lld:lifeskim |
| pubmed-article:9742979 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:9742979 | pubmed:issue | 9131 | lld:pubmed |
| pubmed-article:9742979 | pubmed:dateCreated | 1998-10-1 | lld:pubmed |
| pubmed-article:9742979 | pubmed:abstractText | A polymorphism at codon 72 of the human tumour-suppressor gene, p53, results in translation to either arginine or proline. A recent report suggested that the risk of human-papillomavirus-associated cervical cancer in white women is higher for those homozygous for the arginine allele than for those who are heterozygous. We examined a similar number of cervical cancers and a larger control group for their p53 codon 72 polymorphism status to see if we could confirm this result. | lld:pubmed |
| pubmed-article:9742979 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9742979 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9742979 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9742979 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:9742979 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9742979 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:9742979 | pubmed:issn | 0140-6736 | lld:pubmed |
| pubmed-article:9742979 | pubmed:author | pubmed-author:RyanAA | lld:pubmed |
| pubmed-article:9742979 | pubmed:author | pubmed-author:HarwoodC ACA | lld:pubmed |
| pubmed-article:9742979 | pubmed:author | pubmed-author:StoreyAA | lld:pubmed |
| pubmed-article:9742979 | pubmed:author | pubmed-author:JacobsI JIJ | lld:pubmed |
| pubmed-article:9742979 | pubmed:author | pubmed-author:RosenthalA... | lld:pubmed |
| pubmed-article:9742979 | pubmed:author | pubmed-author:Al-JehaniR... | lld:pubmed |
| pubmed-article:9742979 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9742979 | pubmed:day | 12 | lld:pubmed |
| pubmed-article:9742979 | pubmed:volume | 352 | lld:pubmed |
| pubmed-article:9742979 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9742979 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9742979 | pubmed:pagination | 871-2 | lld:pubmed |
| pubmed-article:9742979 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:9742979 | pubmed:meshHeading | pubmed-meshheading:9742979-... | lld:pubmed |
| pubmed-article:9742979 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9742979 | pubmed:articleTitle | p53 codon 72 polymorphism and risk of cervical cancer in UK. | lld:pubmed |
| pubmed-article:9742979 | pubmed:affiliation | Gynaecology Cancer Research Unit, St Bartholomew's and Royal London School of Medicine and Dentistry, Queen Mary and Westfield College, UK. a.n.rosenthal@mds.qmc.ac.uk | lld:pubmed |
| pubmed-article:9742979 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9742979 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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