| pubmed-article:9696880 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9696880 | lifeskim:mentions | umls-concept:C0013590 | lld:lifeskim |
| pubmed-article:9696880 | lifeskim:mentions | umls-concept:C0042769 | lld:lifeskim |
| pubmed-article:9696880 | lifeskim:mentions | umls-concept:C0020792 | lld:lifeskim |
| pubmed-article:9696880 | lifeskim:mentions | umls-concept:C1708726 | lld:lifeskim |
| pubmed-article:9696880 | lifeskim:mentions | umls-concept:C0132555 | lld:lifeskim |
| pubmed-article:9696880 | lifeskim:mentions | umls-concept:C0683598 | lld:lifeskim |
| pubmed-article:9696880 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:9696880 | pubmed:dateCreated | 1998-9-16 | lld:pubmed |
| pubmed-article:9696880 | pubmed:abstractText | To assess whether nitric oxide synthase 2 (NOS2) fulfills the criteria of an innate resistance locus against an acute viral infection, we inoculated genetically deficient NOS2-/- mice with virulent ectromelia virus (EV), the causative agent of mousepox. NOS2-/- mice proved highly susceptible to EV yet showed no diminution in other well-characterized anti-EV immune responses, i.e. , gamma interferon secretion and NK cell and EV-specific cytotoxic T lymphocyte activities. Thus, the NOS2 locus can be considered a critical monogenic determinant of EV resistance that contributes to host survival. | lld:pubmed |
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| pubmed-article:9696880 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9696880 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9696880 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9696880 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9696880 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:9696880 | pubmed:issn | 0022-538X | lld:pubmed |
| pubmed-article:9696880 | pubmed:author | pubmed-author:ChenJ HJH | lld:pubmed |
| pubmed-article:9696880 | pubmed:author | pubmed-author:NathanC FCF | lld:pubmed |
| pubmed-article:9696880 | pubmed:author | pubmed-author:MacMickingJ... | lld:pubmed |
| pubmed-article:9696880 | pubmed:author | pubmed-author:KarupiahGG | lld:pubmed |
| pubmed-article:9696880 | pubmed:author | pubmed-author:MahalingamSS | lld:pubmed |
| pubmed-article:9696880 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9696880 | pubmed:volume | 72 | lld:pubmed |
| pubmed-article:9696880 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9696880 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9696880 | pubmed:pagination | 7703-6 | lld:pubmed |
| pubmed-article:9696880 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:9696880 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9696880 | pubmed:articleTitle | Identification of nitric oxide synthase 2 as an innate resistance locus against ectromelia virus infection. | lld:pubmed |
| pubmed-article:9696880 | pubmed:affiliation | Host Defense Laboratory, Viral Engineering and Cytokines Group, Division of Immunology and Cell Biology, The John Curtin School of Medical Research, The Australian National University, Canberra, ACT 2601, Australia. Guna.Karupiah@anu.edu.au | lld:pubmed |
| pubmed-article:9696880 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9696880 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:9696880 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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