| pubmed-article:9694807 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9694807 | lifeskim:mentions | umls-concept:C0079427 | lld:lifeskim |
| pubmed-article:9694807 | lifeskim:mentions | umls-concept:C2827404 | lld:lifeskim |
| pubmed-article:9694807 | lifeskim:mentions | umls-concept:C0037083 | lld:lifeskim |
| pubmed-article:9694807 | lifeskim:mentions | umls-concept:C0079419 | lld:lifeskim |
| pubmed-article:9694807 | lifeskim:mentions | umls-concept:C1709313 | lld:lifeskim |
| pubmed-article:9694807 | lifeskim:mentions | umls-concept:C1325410 | lld:lifeskim |
| pubmed-article:9694807 | lifeskim:mentions | umls-concept:C1710082 | lld:lifeskim |
| pubmed-article:9694807 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:9694807 | pubmed:issue | 15 | lld:pubmed |
| pubmed-article:9694807 | pubmed:dateCreated | 1998-8-27 | lld:pubmed |
| pubmed-article:9694807 | pubmed:abstractText | The adenovirus E1A oncogene activates p53 through a signaling pathway involving the retinoblastoma protein and the tumor suppressor p19(ARF). The ability of E1A to induce p53 and its transcriptional targets is severely compromised in ARF-null cells, which remain resistant to apoptosis following serum depletion or adriamycin treatment. Reintroduction of p19(ARF) restores p53 accumulation and resensitizes ARF-null cells to apoptotic signals. Therefore, p19(ARF) functions as part of a p53-dependent failsafe mechanism to counter uncontrolled proliferation. Synergistic effects between the p19(ARF) and DNA damage pathways in inducing p53 may contribute to E1A's ability to enhance radio- and chemosensitivity. | lld:pubmed |
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| pubmed-article:9694807 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9694807 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9694807 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9694807 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9694807 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:9694807 | pubmed:issn | 0890-9369 | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:SherrC JCJ | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:PrivesCC | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:ZindyFF | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:McCurrachM... | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:RousselM FMF | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:ShierS GSG | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:LoweS WSW | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:FerbeyreGG | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:de... | lld:pubmed |
| pubmed-article:9694807 | pubmed:author | pubmed-author:SamuelsonA... | lld:pubmed |
| pubmed-article:9694807 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9694807 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:9694807 | pubmed:volume | 12 | lld:pubmed |
| pubmed-article:9694807 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9694807 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9694807 | pubmed:pagination | 2434-42 | lld:pubmed |
| pubmed-article:9694807 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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