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| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C0032854 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C1883552 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C0226130 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C0021516 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C0598959 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C1707455 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C0178587 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C0205251 | lld:lifeskim |
| pubmed-article:9445353 | lifeskim:mentions | umls-concept:C1561558 | lld:lifeskim |
| pubmed-article:9445353 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:9445353 | pubmed:dateCreated | 1998-2-11 | lld:pubmed |
| pubmed-article:9445353 | pubmed:abstractText | The primary goal of these studies was to understand and investigate the capacity of perivascular nerves to influence the tone of human pial arteries and to compare them with other human cephalic arteries, the superficial temporal and middle meningeal. | lld:pubmed |
| pubmed-article:9445353 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:9445353 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9445353 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9445353 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:9445353 | pubmed:issn | 0039-2499 | lld:pubmed |
| pubmed-article:9445353 | pubmed:author | pubmed-author:BevanJ AJA | lld:pubmed |
| pubmed-article:9445353 | pubmed:author | pubmed-author:BevanR DRD | lld:pubmed |
| pubmed-article:9445353 | pubmed:author | pubmed-author:WaltersC LCL | lld:pubmed |
| pubmed-article:9445353 | pubmed:author | pubmed-author:NicholsPP | lld:pubmed |
| pubmed-article:9445353 | pubmed:author | pubmed-author:DodgeJJ | lld:pubmed |
| pubmed-article:9445353 | pubmed:author | pubmed-author:PenarP LPL | lld:pubmed |
| pubmed-article:9445353 | pubmed:author | pubmed-author:WellmanTT | lld:pubmed |
| pubmed-article:9445353 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9445353 | pubmed:volume | 29 | lld:pubmed |
| pubmed-article:9445353 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9445353 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9445353 | pubmed:pagination | 212-21 | lld:pubmed |
| pubmed-article:9445353 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:9445353 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9445353 | pubmed:articleTitle | Weakness of sympathetic neural control of human pial compared with superficial temporal arteries reflects low innervation density and poor sympathetic responsiveness. | lld:pubmed |
| pubmed-article:9445353 | pubmed:affiliation | Totman Laboratory for Human Cerebrovascular Research, Department of Pharmacology, University of Vermont, Burlington 05405-0068, USA. | lld:pubmed |
| pubmed-article:9445353 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9445353 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:9445353 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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