| pubmed-article:9419206 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0021311 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0877837 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0021368 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0023884 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0128546 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0376579 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C1545588 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C2709199 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C0851827 | lld:lifeskim |
| pubmed-article:9419206 | lifeskim:mentions | umls-concept:C1701901 | lld:lifeskim |
| pubmed-article:9419206 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:9419206 | pubmed:dateCreated | 1998-1-27 | lld:pubmed |
| pubmed-article:9419206 | pubmed:abstractText | Natural killer (NK) cells mediate defense against early murine cytomegalovirus (MCMV) infections in liver. The chemokine, macrophage inflammatory protein 1alpha (MIP-1alpha), can promote inflammatory responses. Our studies evaluated contributions of NK cells to early MCMV-induced liver inflammation and MIP-1alpha requirements for inflammation and delivery of antiviral defenses. NK cells were shown to be responsible for focal inflammation, and to be induced to migrate at high levels, in MCMV-infected livers. MIP-1alpha gene expression was elevated at coinciding times, and mice deficient in MIP-1alpha function were dramatically inhibited in both inflammatory and protective liver responses. The results precisely define MIP-1alpha-dependent steps required to achieve NK cell inflammation during, and mechanisms promoting defense against, viral infections in tissues. | lld:pubmed |
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| pubmed-article:9419206 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9419206 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9419206 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9419206 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9419206 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:9419206 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:9419206 | pubmed:author | pubmed-author:BironC ACA | lld:pubmed |
| pubmed-article:9419206 | pubmed:author | pubmed-author:OrangeJ SJS | lld:pubmed |
| pubmed-article:9419206 | pubmed:author | pubmed-author:Salazar-Mathe... | lld:pubmed |
| pubmed-article:9419206 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9419206 | pubmed:day | 5 | lld:pubmed |
| pubmed-article:9419206 | pubmed:volume | 187 | lld:pubmed |
| pubmed-article:9419206 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9419206 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9419206 | pubmed:pagination | 1-14 | lld:pubmed |
| pubmed-article:9419206 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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