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| pubmed-article:9393765 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:9393765 | lifeskim:mentions | umls-concept:C0017638 | lld:lifeskim |
| pubmed-article:9393765 | lifeskim:mentions | umls-concept:C0025646 | lld:lifeskim |
| pubmed-article:9393765 | lifeskim:mentions | umls-concept:C0062534 | lld:lifeskim |
| pubmed-article:9393765 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:9393765 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:9393765 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:9393765 | pubmed:issue | 23 | lld:pubmed |
| pubmed-article:9393765 | pubmed:dateCreated | 1998-3-2 | lld:pubmed |
| pubmed-article:9393765 | pubmed:abstractText | Using double immunofluorescence staining and quantitative confocal laser scan microscopy, we show that the intensity of hepatocyte growth factor/scatter factor (HGF/SF) and Met staining in human primary brain tumors increases with the grade of malignancy and is prevalent in both the infiltrating tumor cells and endothelial hyperplastic areas. HGF/SF and Met also are expressed in vitro in glioblastoma multiforme cell lines as well as in normal human astrocyte (NHA) cells. Moreover, HGF/SF stimulates tyrosine phosphorylation of Met in both glioma cell lines and NHA cells, but only the glioma cell lines proliferate and become motile and invasive in response to HGF/SF, whereas the NHA cells are nonresponsive. These results implicate autocrine/paracrine Met-HGF/SF signaling in glioma tumorigenesis and suggest that HGF/SF signaling through Met is negatively regulated in NHA cells. | lld:pubmed |
| pubmed-article:9393765 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9393765 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9393765 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9393765 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9393765 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:9393765 | pubmed:issn | 0008-5472 | lld:pubmed |
| pubmed-article:9393765 | pubmed:author | pubmed-author:TaylorGG | lld:pubmed |
| pubmed-article:9393765 | pubmed:author | pubmed-author:Vande... | lld:pubmed |
| pubmed-article:9393765 | pubmed:author | pubmed-author:HudsonE AEA | lld:pubmed |
| pubmed-article:9393765 | pubmed:author | pubmed-author:ResauJ HJH | lld:pubmed |
| pubmed-article:9393765 | pubmed:author | pubmed-author:RulongSS | lld:pubmed |
| pubmed-article:9393765 | pubmed:author | pubmed-author:JefferyWW | lld:pubmed |
| pubmed-article:9393765 | pubmed:author | pubmed-author:KoochekpourSS | lld:pubmed |
| pubmed-article:9393765 | pubmed:author | pubmed-author:KlinebergEE | lld:pubmed |
| pubmed-article:9393765 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9393765 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:9393765 | pubmed:volume | 57 | lld:pubmed |
| pubmed-article:9393765 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9393765 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9393765 | pubmed:pagination | 5391-8 | lld:pubmed |
| pubmed-article:9393765 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:9393765 | pubmed:year | 1997 | lld:pubmed |
| pubmed-article:9393765 | pubmed:articleTitle | Met and hepatocyte growth factor/scatter factor expression in human gliomas. | lld:pubmed |
| pubmed-article:9393765 | pubmed:affiliation | ABL-Basic Research Program, National Cancer Institute, Frederick Cancer Research and Development Center, Maryland 21702, USA. | lld:pubmed |
| pubmed-article:9393765 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9393765 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:9393765 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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