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| pubmed-article:9337776 | lifeskim:mentions | umls-concept:C0470187 | lld:lifeskim |
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| pubmed-article:9337776 | lifeskim:mentions | umls-concept:C0205099 | lld:lifeskim |
| pubmed-article:9337776 | lifeskim:mentions | umls-concept:C0205421 | lld:lifeskim |
| pubmed-article:9337776 | lifeskim:mentions | umls-concept:C1882115 | lld:lifeskim |
| pubmed-article:9337776 | lifeskim:mentions | umls-concept:C0439611 | lld:lifeskim |
| pubmed-article:9337776 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:9337776 | pubmed:dateCreated | 1997-11-4 | lld:pubmed |
| pubmed-article:9337776 | pubmed:abstractText | Antipsychotic drugs in depot formulations may prevent psychotic relapses, even after complete withdrawal. To examine the duration of drug remaining in the brain, central D2 dopamine receptor occupancy was measured with positron emission tomography for a year after discontinuation of depot neuroleptic treatment. | lld:pubmed |
| pubmed-article:9337776 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9337776 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9337776 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9337776 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:9337776 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9337776 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:9337776 | pubmed:issn | 0003-990X | lld:pubmed |
| pubmed-article:9337776 | pubmed:author | pubmed-author:FardeLL | lld:pubmed |
| pubmed-article:9337776 | pubmed:author | pubmed-author:HalldinCC | lld:pubmed |
| pubmed-article:9337776 | pubmed:author | pubmed-author:NybergSS | lld:pubmed |
| pubmed-article:9337776 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9337776 | pubmed:volume | 54 | lld:pubmed |
| pubmed-article:9337776 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9337776 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9337776 | pubmed:pagination | 953-8 | lld:pubmed |
| pubmed-article:9337776 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:9337776 | pubmed:year | 1997 | lld:pubmed |
| pubmed-article:9337776 | pubmed:articleTitle | Delayed normalization of central D2 dopamine receptor availability after discontinuation of haloperidol decanoate. Preliminary findings. | lld:pubmed |
| pubmed-article:9337776 | pubmed:affiliation | Department of Clinical Neuroscience, Karolinska Institutet, Karolinska Hospital, Stockholm, Sweden. svanten@psyk.ks.se | lld:pubmed |
| pubmed-article:9337776 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9337776 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:9337776 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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