| pubmed-article:9334375 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C1123023 | lld:lifeskim |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C0024109 | lld:lifeskim |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C0031153 | lld:lifeskim |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C0027950 | lld:lifeskim |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C0064830 | lld:lifeskim |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C0054942 | lld:lifeskim |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C0013975 | lld:lifeskim |
| pubmed-article:9334375 | lifeskim:mentions | umls-concept:C1514873 | lld:lifeskim |
| pubmed-article:9334375 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:9334375 | pubmed:dateCreated | 1997-11-21 | lld:pubmed |
| pubmed-article:9334375 | pubmed:abstractText | To determine the role of CD11/CD18 complexes in neutrophil emigration, inflammation was induced in the skin, lungs, or peritoneum of mutant mice deficient in CD18 (CD18-/- mutants). Peripheral blood of CD18-/- mutants contained 11-fold more neutrophils than did blood of wild-type (WT) mice. During irritant dermatitis induced by topical application of croton oil, the number of emigrated neutrophils in histological sections of dermis was 98% less in CD18-/- mutants than in WT mice. During Streptococcus pneumoniae pneumonia, neutrophil emigration in CD18-/- mutants was not reduced. These data are consistent with expectations based on studies using blocking antibodies to inhibit CD11/CD18 complexes, and on observations of humans lacking CD11/CD18 complexes. The number of emigrated neutrophils in lung sections during Escherichia coli pneumonia, or in peritoneal lavage fluid after 4 h of S. pneumoniae peritonitis, was not reduced in CD18-/- mutants, but rather was greater than the WT values (240 +/- 30 and 220 +/- 30% WT, respectively). Also, there was no inhibition of neutrophil emigration during sterile peritonitis induced by intraperitoneal injection of thioglycollate (90 +/- 20% WT). These data contrast with expectations. Whereas CD11/CD18 complexes are essential to the dermal emigration of neutrophils during acute dermatitis, CD18-/- mutant mice demonstrate surprising alternative pathways for neutrophil emigration during pneumonia or peritonitis. | lld:pubmed |
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| pubmed-article:9334375 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:9334375 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9334375 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9334375 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:9334375 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:9334375 | pubmed:author | pubmed-author:KuboHH | lld:pubmed |
| pubmed-article:9334375 | pubmed:author | pubmed-author:BeaudetA LAL | lld:pubmed |
| pubmed-article:9334375 | pubmed:author | pubmed-author:DoerschukC... | lld:pubmed |
| pubmed-article:9334375 | pubmed:author | pubmed-author:Scharffetter-... | lld:pubmed |
| pubmed-article:9334375 | pubmed:author | pubmed-author:MizgerdJ PJP | lld:pubmed |
| pubmed-article:9334375 | pubmed:author | pubmed-author:KutkoskiG JGJ | lld:pubmed |
| pubmed-article:9334375 | pubmed:author | pubmed-author:BhagwanS DSD | lld:pubmed |
| pubmed-article:9334375 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9334375 | pubmed:day | 20 | lld:pubmed |
| pubmed-article:9334375 | pubmed:volume | 186 | lld:pubmed |
| pubmed-article:9334375 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9334375 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9334375 | pubmed:pagination | 1357-64 | lld:pubmed |
| pubmed-article:9334375 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:9334375 | pubmed:year | 1997 | lld:pubmed |
| pubmed-article:9334375 | pubmed:articleTitle | Neutrophil emigration in the skin, lungs, and peritoneum: different requirements for CD11/CD18 revealed by CD18-deficient mice. | lld:pubmed |
| pubmed-article:9334375 | pubmed:affiliation | Physiology Program, Harvard School of Public Health, Boston, Massachusetts 02115, USA. | lld:pubmed |
| pubmed-article:9334375 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9334375 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:9334375 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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