| pubmed-article:9252126 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C1257890 | lld:lifeskim |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C0024660 | lld:lifeskim |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C0024264 | lld:lifeskim |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C1538598 | lld:lifeskim |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C0083956 | lld:lifeskim |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C1514485 | lld:lifeskim |
| pubmed-article:9252126 | lifeskim:mentions | umls-concept:C1709634 | lld:lifeskim |
| pubmed-article:9252126 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:9252126 | pubmed:dateCreated | 1997-9-4 | lld:pubmed |
| pubmed-article:9252126 | pubmed:abstractText | mel-18 is a mammalian homolog of Drosophila melanogaster Polycomb group genes. Mice lacking the mel-18 gene show a posterior transformation of the axial skeleton, severe combined immunodeficiency, and a food-passing disturbance in the lower intestine due to hypertrophy of the smooth muscle layer. In this study, the severe combined immunodeficiency observed in mel-18 mutant mice is correlated with the impaired mitotic response of lymphocyte precursors upon interleukin-7 stimulation. Strikingly, the axial skeleton and lymphoid phenotypes are identical in both mel-18 and bmi-1 mutants, indicating that the Mel-18 and Bmi-1 gene products might act in the same genetic cascade. These results suggest that mammalian Polycomb group gene products are involved in cell cycle progression in the immune system. | lld:pubmed |
| pubmed-article:9252126 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9252126 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9252126 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:9252126 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9252126 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9252126 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9252126 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9252126 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:9252126 | pubmed:issn | 1074-7613 | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:KannoMM | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:TsujiKK | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:TaniguchiMM | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:HarigayaKK | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:NakahataTT | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:EbiharaYY | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:AkasakaTT | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:IIAA | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:KosekiHH | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:TetsuOO | lld:pubmed |
| pubmed-article:9252126 | pubmed:author | pubmed-author:KawahiraHH | lld:pubmed |
| pubmed-article:9252126 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9252126 | pubmed:volume | 7 | lld:pubmed |
| pubmed-article:9252126 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9252126 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9252126 | pubmed:pagination | 135-46 | lld:pubmed |
| pubmed-article:9252126 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:9252126 | pubmed:meshHeading | pubmed-meshheading:9252126-... | lld:pubmed |
| pubmed-article:9252126 | pubmed:meshHeading | pubmed-meshheading:9252126-... | lld:pubmed |
| pubmed-article:9252126 | pubmed:meshHeading | pubmed-meshheading:9252126-... | lld:pubmed |
| pubmed-article:9252126 | pubmed:year | 1997 | lld:pubmed |
| pubmed-article:9252126 | pubmed:articleTitle | The role of mel-18, a mammalian Polycomb group gene, during IL-7-dependent proliferation of lymphocyte precursors. | lld:pubmed |
| pubmed-article:9252126 | pubmed:affiliation | Core Research for Evolution Science and Technology, Japan Science and Technology Corporation, and Division of Molecular Immunology, Center for Biomedical Science, School of Medicine, Chiba University. | lld:pubmed |
| pubmed-article:9252126 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9252126 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:22658 | entrezgene:pubmed | pubmed-article:9252126 | lld:entrezgene |
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