pubmed-article:9207249 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:9207249 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:9207249 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
pubmed-article:9207249 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
pubmed-article:9207249 | lifeskim:mentions | umls-concept:C0521390 | lld:lifeskim |
pubmed-article:9207249 | lifeskim:mentions | umls-concept:C0085151 | lld:lifeskim |
pubmed-article:9207249 | lifeskim:mentions | umls-concept:C1709694 | lld:lifeskim |
pubmed-article:9207249 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:9207249 | pubmed:dateCreated | 1997-7-24 | lld:pubmed |
pubmed-article:9207249 | pubmed:abstractText | The processing of Alzheimer's amyloid precursor protein was studied by Western blotting during H2O2 induced apoptosis in cultures of human neuroblastoma cells. A new 5.5 kDa fragment putatively containing intact A beta was detected and found to be highly associated with apoptosis. The results suggest a possible vicious cycle involving H2O2, A beta and apoptosis which may contribute to the neuronal death mechanism in Alzheimer's Disease. | lld:pubmed |
pubmed-article:9207249 | pubmed:language | eng | lld:pubmed |
pubmed-article:9207249 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9207249 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:9207249 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9207249 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9207249 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9207249 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9207249 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:9207249 | pubmed:month | Jun | lld:pubmed |
pubmed-article:9207249 | pubmed:issn | 0006-291X | lld:pubmed |
pubmed-article:9207249 | pubmed:author | pubmed-author:KusiakJ WJW | lld:pubmed |
pubmed-article:9207249 | pubmed:author | pubmed-author:YewD TDT | lld:pubmed |
pubmed-article:9207249 | pubmed:author | pubmed-author:RothG SGS | lld:pubmed |
pubmed-article:9207249 | pubmed:author | pubmed-author:ZhangLL | lld:pubmed |
pubmed-article:9207249 | pubmed:author | pubmed-author:ZhaoBB | lld:pubmed |
pubmed-article:9207249 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:9207249 | pubmed:day | 27 | lld:pubmed |
pubmed-article:9207249 | pubmed:volume | 235 | lld:pubmed |
pubmed-article:9207249 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:9207249 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:9207249 | pubmed:pagination | 845-8 | lld:pubmed |
pubmed-article:9207249 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:9207249 | pubmed:meshHeading | pubmed-meshheading:9207249-... | lld:pubmed |
pubmed-article:9207249 | pubmed:year | 1997 | lld:pubmed |
pubmed-article:9207249 | pubmed:articleTitle | Processing of Alzheimer's amyloid precursor protein during H2O2-induced apoptosis in human neuronal cells. | lld:pubmed |
pubmed-article:9207249 | pubmed:affiliation | Laboratory of Cellular and Molecular Biology, Gerontology Research Center, NIA/NIH, Baltimore, Maryland, USA. | lld:pubmed |
pubmed-article:9207249 | pubmed:publicationType | Journal Article | lld:pubmed |
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