| pubmed-article:9151703 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9151703 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:9151703 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:9151703 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:9151703 | lifeskim:mentions | umls-concept:C0258439 | lld:lifeskim |
| pubmed-article:9151703 | lifeskim:mentions | umls-concept:C1420887 | lld:lifeskim |
| pubmed-article:9151703 | lifeskim:mentions | umls-concept:C0220905 | lld:lifeskim |
| pubmed-article:9151703 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:9151703 | pubmed:dateCreated | 1997-6-24 | lld:pubmed |
| pubmed-article:9151703 | pubmed:abstractText | Tumor necrosis factor receptor (TNFR)-associated factor 2 (TRAF2) and TRAF1 were found as components of the TNFR2 signaling complex, which exerts multiple biological effects on cells such as cell proliferation, cytokine production, and cell death. In the TNFR2-mediated signaling pathways, TRAF2 works as a mediator for activation signals such as NF-kappaB, but the role of TRAF1 has not been previously determined. Here we show in transgenic mice that TRAF1 overexpression inhibits antigen-induced apoptosis of CD8(+) T lymphocytes. Our results demonstrate a biological role for TRAF1 as a regulator of apoptotic signals and also support the hypothesis that the combination of TRAF proteins in a given cell type determines distinct biological effects triggered by members of the TNF receptor superfamily. | lld:pubmed |
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| pubmed-article:9151703 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9151703 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9151703 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:9151703 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9151703 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9151703 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9151703 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9151703 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:9151703 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9151703 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9151703 | pubmed:month | May | lld:pubmed |
| pubmed-article:9151703 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:9151703 | pubmed:author | pubmed-author:LeeS YSY | lld:pubmed |
| pubmed-article:9151703 | pubmed:author | pubmed-author:WongBB | lld:pubmed |
| pubmed-article:9151703 | pubmed:author | pubmed-author:ChoiYY | lld:pubmed |
| pubmed-article:9151703 | pubmed:author | pubmed-author:SpeiserD EDE | lld:pubmed |
| pubmed-article:9151703 | pubmed:author | pubmed-author:SantanaAA | lld:pubmed |
| pubmed-article:9151703 | pubmed:author | pubmed-author:OhashiP SPS | lld:pubmed |
| pubmed-article:9151703 | pubmed:author | pubmed-author:KongY YYY | lld:pubmed |
| pubmed-article:9151703 | pubmed:author | pubmed-author:ArronJJ | lld:pubmed |
| pubmed-article:9151703 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9151703 | pubmed:day | 19 | lld:pubmed |
| pubmed-article:9151703 | pubmed:volume | 185 | lld:pubmed |
| pubmed-article:9151703 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9151703 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9151703 | pubmed:pagination | 1777-83 | lld:pubmed |
| pubmed-article:9151703 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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