| pubmed-article:9138902 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9138902 | lifeskim:mentions | umls-concept:C0002312 | lld:lifeskim |
| pubmed-article:9138902 | lifeskim:mentions | umls-concept:C2707017 | lld:lifeskim |
| pubmed-article:9138902 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
| pubmed-article:9138902 | lifeskim:mentions | umls-concept:C0348080 | lld:lifeskim |
| pubmed-article:9138902 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:9138902 | pubmed:dateCreated | 1997-5-2 | lld:pubmed |
| pubmed-article:9138902 | pubmed:abstractText | Mild alpha thalassemia is the most prevalent genetic trait worldwide. We have recently developed a multiplex polymerase chain reaction method to screen for the most common deletions which give rise to alpha thalassemia. The authors have used this method to investigate a potential association of alpha thalassemia with hypertension. Our results show that the prevalence of hypertension in hospitalized blacks who have the -alpha 3.7 deletion is 71 percent higher than the prevalence in hospitalized blacks who do not have the deletion. The authors present a potential mechanism to explain this association. | lld:pubmed |
| pubmed-article:9138902 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9138902 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9138902 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:9138902 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9138902 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9138902 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9138902 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:9138902 | pubmed:issn | 0272-2712 | lld:pubmed |
| pubmed-article:9138902 | pubmed:author | pubmed-author:BowieL JLJ | lld:pubmed |
| pubmed-article:9138902 | pubmed:author | pubmed-author:ReddyP LPL | lld:pubmed |
| pubmed-article:9138902 | pubmed:author | pubmed-author:BeckK RKR | lld:pubmed |
| pubmed-article:9138902 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9138902 | pubmed:volume | 17 | lld:pubmed |
| pubmed-article:9138902 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9138902 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9138902 | pubmed:pagination | 97-108 | lld:pubmed |
| pubmed-article:9138902 | pubmed:dateRevised | 2006-3-28 | lld:pubmed |
| pubmed-article:9138902 | pubmed:meshHeading | pubmed-meshheading:9138902-... | lld:pubmed |
| pubmed-article:9138902 | pubmed:meshHeading | pubmed-meshheading:9138902-... | lld:pubmed |
| pubmed-article:9138902 | pubmed:meshHeading | pubmed-meshheading:9138902-... | lld:pubmed |
| pubmed-article:9138902 | pubmed:meshHeading | pubmed-meshheading:9138902-... | lld:pubmed |
| pubmed-article:9138902 | pubmed:meshHeading | pubmed-meshheading:9138902-... | lld:pubmed |
| pubmed-article:9138902 | pubmed:meshHeading | pubmed-meshheading:9138902-... | lld:pubmed |
| pubmed-article:9138902 | pubmed:year | 1997 | lld:pubmed |
| pubmed-article:9138902 | pubmed:articleTitle | Alpha thalassemia and its impact on other clinical conditions. | lld:pubmed |
| pubmed-article:9138902 | pubmed:affiliation | Department of Pathology, Northwestern University Medical School, Chicago, USA. | lld:pubmed |
| pubmed-article:9138902 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9138902 | pubmed:publicationType | Review | lld:pubmed |
