| pubmed-article:8828436 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8828436 | lifeskim:mentions | umls-concept:C0043210 | lld:lifeskim |
| pubmed-article:8828436 | lifeskim:mentions | umls-concept:C0040679 | lld:lifeskim |
| pubmed-article:8828436 | lifeskim:mentions | umls-concept:C0229671 | lld:lifeskim |
| pubmed-article:8828436 | lifeskim:mentions | umls-concept:C0242606 | lld:lifeskim |
| pubmed-article:8828436 | lifeskim:mentions | umls-concept:C0032914 | lld:lifeskim |
| pubmed-article:8828436 | lifeskim:mentions | umls-concept:C0919786 | lld:lifeskim |
| pubmed-article:8828436 | lifeskim:mentions | umls-concept:C0205216 | lld:lifeskim |
| pubmed-article:8828436 | pubmed:issue | 3 Pt 1 | lld:pubmed |
| pubmed-article:8828436 | pubmed:dateCreated | 1996-11-4 | lld:pubmed |
| pubmed-article:8828436 | pubmed:abstractText | The concerted iron-binding antioxidant activity of transferrin and ceruloplasmin decreases with increasing transferrin saturation by iron. We examined interactions between serum iron and iron-binding capacity and concentrations of the lipid peroxidation metabolite malondialdehyde in normal and preeclamptic pregnancies. We also asked if the release of iron from free hemoglobin by lipid hydroperoxides is a potential mechanism to increase transferrin saturation in preeclampsia. | lld:pubmed |
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| pubmed-article:8828436 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8828436 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:8828436 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8828436 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:8828436 | pubmed:issn | 0002-9378 | lld:pubmed |
| pubmed-article:8828436 | pubmed:author | pubmed-author:RobertsJ MJM | lld:pubmed |
| pubmed-article:8828436 | pubmed:author | pubmed-author:McLaughlinM... | lld:pubmed |
| pubmed-article:8828436 | pubmed:author | pubmed-author:KaganV EVE | lld:pubmed |
| pubmed-article:8828436 | pubmed:author | pubmed-author:KozlovA VAV | lld:pubmed |
| pubmed-article:8828436 | pubmed:author | pubmed-author:EvansR WRW | lld:pubmed |
| pubmed-article:8828436 | pubmed:author | pubmed-author:DavidgeS TST | lld:pubmed |
| pubmed-article:8828436 | pubmed:author | pubmed-author:HubelC ACA | lld:pubmed |
| pubmed-article:8828436 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8828436 | pubmed:volume | 175 | lld:pubmed |
| pubmed-article:8828436 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8828436 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8828436 | pubmed:pagination | 692-700 | lld:pubmed |
| pubmed-article:8828436 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:8828436 | pubmed:meshHeading | pubmed-meshheading:8828436-... | lld:pubmed |
| pubmed-article:8828436 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8828436 | pubmed:articleTitle | Decreased transferrin and increased transferrin saturation in sera of women with preeclampsia: implications for oxidative stress. | lld:pubmed |
| pubmed-article:8828436 | pubmed:affiliation | Magee-Womens Research Institute, Pittsburgh, PA 15213, USA. | lld:pubmed |
| pubmed-article:8828436 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8828436 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8828436 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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