pubmed-article:8823339 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C0032854 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C0278493 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C0034804 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C0279025 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C1561558 | lld:lifeskim |
pubmed-article:8823339 | lifeskim:mentions | umls-concept:C1517945 | lld:lifeskim |
pubmed-article:8823339 | pubmed:issue | 9 | lld:pubmed |
pubmed-article:8823339 | pubmed:dateCreated | 1996-10-30 | lld:pubmed |
pubmed-article:8823339 | pubmed:abstractText | Up to 30% to 40% of metastases from hormone receptor-positive primary breast cancer do not respond to endocrine therapy. We studied how often hormone receptor status changes between primary and recurrent tumors and whether such a change might explain unresponsiveness to endocrine therapy. | lld:pubmed |
pubmed-article:8823339 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8823339 | pubmed:language | eng | lld:pubmed |
pubmed-article:8823339 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8823339 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:8823339 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:8823339 | pubmed:month | Sep | lld:pubmed |
pubmed-article:8823339 | pubmed:issn | 0732-183X | lld:pubmed |
pubmed-article:8823339 | pubmed:author | pubmed-author:HolmeTT | lld:pubmed |
pubmed-article:8823339 | pubmed:author | pubmed-author:HelinHH | lld:pubmed |
pubmed-article:8823339 | pubmed:author | pubmed-author:KononenJJ | lld:pubmed |
pubmed-article:8823339 | pubmed:author | pubmed-author:IsolaJJ | lld:pubmed |
pubmed-article:8823339 | pubmed:author | pubmed-author:KuukasjärviTT | lld:pubmed |
pubmed-article:8823339 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:8823339 | pubmed:volume | 14 | lld:pubmed |
pubmed-article:8823339 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:8823339 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:8823339 | pubmed:pagination | 2584-9 | lld:pubmed |
pubmed-article:8823339 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:8823339 | pubmed:year | 1996 | lld:pubmed |
pubmed-article:8823339 | pubmed:articleTitle | Loss of estrogen receptor in recurrent breast cancer is associated with poor response to endocrine therapy. | lld:pubmed |
pubmed-article:8823339 | pubmed:affiliation | Department of Pathology, Tampere University Hospital, University of Tampere, Finland. bltuku@uta.ft | lld:pubmed |
pubmed-article:8823339 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:8823339 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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