| pubmed-article:8815199 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C0034721 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C0016030 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C0068635 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C0596235 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C1698986 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C1948027 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C1550548 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C1555714 | lld:lifeskim |
| pubmed-article:8815199 | lifeskim:mentions | umls-concept:C1705654 | lld:lifeskim |
| pubmed-article:8815199 | pubmed:dateCreated | 1996-10-4 | lld:pubmed |
| pubmed-article:8815199 | pubmed:abstractText | 1. Transfected Chinese hamster ovary fibroblasts expressing the rat neurotensin receptor were used to study the 'Ca2+ stores depletion-Ca2+ entry coupling' which follows stimulation with neurotensin and liberation of InsP3. 2. This coupling could be dissociated in time. Firstly, stores depletion was produced by neurotensin or thapsigargin which caused a first [Ca2+]i transient in a Ca(2+)-free external medium. Secondly, readmission of external Ca2+ produced an influx of Ca2+ and a second [Ca2+]i transient. 3. Various concentrations of thapsigargin (20 nM to 1 microM) were used to produce complete stores depletion with small or large first peaks of [Ca2+]i. Upon return to external Ca2+, small or large second [Ca2+]i peaks were observed. The amplitudes of both peaks were positively correlated. 4. The Ca2+ entry which followed stores depletion could occur at very low basal values of [Ca2+]i, was accelerated by okadaic acid and inhibited by staurosporine and the calmodulin antagonist W-7. 5. It is concluded that the rise in [Ca2+]i during Ca2+ stores depletion is an essential parameter which determines the size of the subsequent Ca2+ entry. | lld:pubmed |
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| pubmed-article:8815199 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8815199 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8815199 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8815199 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:8815199 | pubmed:issn | 0022-3751 | lld:pubmed |
| pubmed-article:8815199 | pubmed:author | pubmed-author:HermannBB | lld:pubmed |
| pubmed-article:8815199 | pubmed:author | pubmed-author:GillisJ MJM | lld:pubmed |
| pubmed-article:8815199 | pubmed:author | pubmed-author:GaillyPP | lld:pubmed |
| pubmed-article:8815199 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8815199 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:8815199 | pubmed:volume | 491 ( Pt 3) | lld:pubmed |
| pubmed-article:8815199 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8815199 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8815199 | pubmed:pagination | 635-46 | lld:pubmed |
| pubmed-article:8815199 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:8815199 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8815199 | pubmed:articleTitle | Role of [Ca2+]i in "Ca2+ stores depletion-Ca2+ entry coupling' in fibroblasts expressing the rat neurotensin receptor. | lld:pubmed |
| pubmed-article:8815199 | pubmed:affiliation | Department of Physiology, Catholic University of Louvain, Brussels, Belgium. | lld:pubmed |
| pubmed-article:8815199 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8815199 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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