| pubmed-article:8796888 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8796888 | lifeskim:mentions | umls-concept:C0012634 | lld:lifeskim |
| pubmed-article:8796888 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:8796888 | lifeskim:mentions | umls-concept:C0026473 | lld:lifeskim |
| pubmed-article:8796888 | lifeskim:mentions | umls-concept:C0282566 | lld:lifeskim |
| pubmed-article:8796888 | lifeskim:mentions | umls-concept:C0271510 | lld:lifeskim |
| pubmed-article:8796888 | lifeskim:mentions | umls-concept:C0333348 | lld:lifeskim |
| pubmed-article:8796888 | lifeskim:mentions | umls-concept:C1704735 | lld:lifeskim |
| pubmed-article:8796888 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:8796888 | pubmed:dateCreated | 1996-10-16 | lld:pubmed |
| pubmed-article:8796888 | pubmed:abstractText | The appearance of specific types of leukocytes in inflammatory infiltrates may be governed by cell-specific chemoattractants called chemokines. In particular, monocyte chemoattractant protein 1 (MCP-1) has been implicated in diseases characterized by monocyte-rich infiltrates, including atherosclerosis, rheumatoid arthritis and multiple sclerosis. While we are beginning to understand the structural determinants that govern the activities of MCP-1 in vitro, we know much less about its physiological functions in vivo and its pathogenetic role in disease. However, recent data from genetically modified mice have begun to place MCP-1 in a central position in monocyte trafficking and activation. | lld:pubmed |
| pubmed-article:8796888 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8796888 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8796888 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:8796888 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8796888 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8796888 | pubmed:month | May | lld:pubmed |
| pubmed-article:8796888 | pubmed:issn | 1357-4310 | lld:pubmed |
| pubmed-article:8796888 | pubmed:author | pubmed-author:RollinsB JBJ | lld:pubmed |
| pubmed-article:8796888 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8796888 | pubmed:volume | 2 | lld:pubmed |
| pubmed-article:8796888 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8796888 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8796888 | pubmed:pagination | 198-204 | lld:pubmed |
| pubmed-article:8796888 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:meshHeading | pubmed-meshheading:8796888-... | lld:pubmed |
| pubmed-article:8796888 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8796888 | pubmed:articleTitle | Monocyte chemoattractant protein 1: a potential regulator of monocyte recruitment in inflammatory disease. | lld:pubmed |
| pubmed-article:8796888 | pubmed:affiliation | Dana-Farber Cancer Institute, Harvard Medical School, Boston, MA 02115, USA. barrett-rollins@macmailgw.dfci.harvard.edu | lld:pubmed |
| pubmed-article:8796888 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8796888 | pubmed:publicationType | Review | lld:pubmed |
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