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pubmed-article:8771501pubmed:abstractTextVascular dysfunction and myocardial contracture can both contribute to posttransplantation diastolic abnormalities commonly exhibited by heart transplants, but their respective importance remains incompletely elucidated. To address this issue, we assessed the effects of supplementing a new heart preservation solution, Celsior, with a nitric oxide precursor (L-arginine) and a compound known to uncouple excitation from contraction (2, 3-butanedione monoxime).lld:pubmed
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pubmed-article:8771501pubmed:articleTitlePoststorage diastolic abnormalities of heart transplants: is vascular dysfunction or myocardial contracture the culprit?lld:pubmed
pubmed-article:8771501pubmed:affiliationDepartment of Physiology, Medical Academy, Kaunas, Lithuania.lld:pubmed
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