| pubmed-article:8760871 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8760871 | lifeskim:mentions | umls-concept:C0039644 | lld:lifeskim |
| pubmed-article:8760871 | lifeskim:mentions | umls-concept:C0017373 | lld:lifeskim |
| pubmed-article:8760871 | lifeskim:mentions | umls-concept:C1336789 | lld:lifeskim |
| pubmed-article:8760871 | lifeskim:mentions | umls-concept:C0231491 | lld:lifeskim |
| pubmed-article:8760871 | lifeskim:mentions | umls-concept:C0679622 | lld:lifeskim |
| pubmed-article:8760871 | lifeskim:mentions | umls-concept:C0205314 | lld:lifeskim |
| pubmed-article:8760871 | pubmed:issue | 15 | lld:pubmed |
| pubmed-article:8760871 | pubmed:dateCreated | 1996-10-10 | lld:pubmed |
| pubmed-article:8760871 | pubmed:abstractText | We have screened a panel of tetracycline (tc)-like compounds for their potential use with tc-repressor (tetR) based gene switches. The interaction between tc and tetR appears quite specific, as only tc itself and its close homologues anhydro-tc and doxycycline strongly inhibited DNA binding. However, a single tc-like compound, GR33076X, increased DNA binding of the tetR-VP16 fusion protein, both in eukaryotic cells and in bacteria. We provide evidence that this antagonist of tetracycline is potentially useful for accelerated gene switching, especially in whole animals. | lld:pubmed |
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| pubmed-article:8760871 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8760871 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8760871 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8760871 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:8760871 | pubmed:issn | 0305-1048 | lld:pubmed |
| pubmed-article:8760871 | pubmed:author | pubmed-author:Hooft van... | lld:pubmed |
| pubmed-article:8760871 | pubmed:author | pubmed-author:Chrast-BalzJJ | lld:pubmed |
| pubmed-article:8760871 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8760871 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:8760871 | pubmed:volume | 24 | lld:pubmed |
| pubmed-article:8760871 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8760871 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8760871 | pubmed:pagination | 2900-4 | lld:pubmed |
| pubmed-article:8760871 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:8760871 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8760871 | pubmed:articleTitle | Bi-directional gene switching with the tetracycline repressor and a novel tetracycline antagonist. | lld:pubmed |
| pubmed-article:8760871 | pubmed:affiliation | Geneva Biomedical Research Institute, Geneva, Switzerland. | lld:pubmed |
| pubmed-article:8760871 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8760871 | pubmed:publicationType | Comparative Study | lld:pubmed |
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