| pubmed-article:8657567 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8657567 | lifeskim:mentions | umls-concept:C0032136 | lld:lifeskim |
| pubmed-article:8657567 | lifeskim:mentions | umls-concept:C0205147 | lld:lifeskim |
| pubmed-article:8657567 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
| pubmed-article:8657567 | lifeskim:mentions | umls-concept:C0242961 | lld:lifeskim |
| pubmed-article:8657567 | lifeskim:mentions | umls-concept:C1704675 | lld:lifeskim |
| pubmed-article:8657567 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:8657567 | pubmed:dateCreated | 1996-7-31 | lld:pubmed |
| pubmed-article:8657567 | pubmed:abstractText | A set of AT-rich repeats is a common motif in prokaryotic replication origins. We have screened for proteins binding to the AT-rich repeat region in plasmids F, R1 and pSC101 using an electrophoretic mobility shift assay with PCR-amplified DNA fragments from the origins. The IciA protein, which is known to bind to the AT-rich repeat region in the Escherichia coli origin of chromosome replication, oriC, was found to bind to the corresponding region from plasmids F (oriS) and R1, but not to pSC101. DNase I footprint analysis showed that IciA interacted with the AT-rich region in both F and R1. When the IciA gene was deleted, the copy number of plasmid F increased somewhat, whereas there was no major effect on the replication of pSC101 and R1, or on the E. coli chromosome. | lld:pubmed |
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| pubmed-article:8657567 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8657567 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8657567 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8657567 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8657567 | pubmed:month | May | lld:pubmed |
| pubmed-article:8657567 | pubmed:issn | 0305-1048 | lld:pubmed |
| pubmed-article:8657567 | pubmed:author | pubmed-author:WeiTT | lld:pubmed |
| pubmed-article:8657567 | pubmed:author | pubmed-author:BernanderRR | lld:pubmed |
| pubmed-article:8657567 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8657567 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:8657567 | pubmed:volume | 24 | lld:pubmed |
| pubmed-article:8657567 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8657567 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8657567 | pubmed:pagination | 1865-72 | lld:pubmed |
| pubmed-article:8657567 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:8657567 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8657567 | pubmed:articleTitle | Interaction of the IciA protein with AT-rich regions in plasmid replication origins. | lld:pubmed |
| pubmed-article:8657567 | pubmed:affiliation | Department of Microbiology, Biomedical Center, Uppsala University, Sweden. | lld:pubmed |
| pubmed-article:8657567 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8657567 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:944867 | entrezgene:pubmed | pubmed-article:8657567 | lld:entrezgene |
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