| pubmed-article:8598453 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C0431085 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C0245662 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C0037083 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C1539477 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C1705955 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C1514562 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C1883221 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C1710082 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C1883204 | lld:lifeskim |
| pubmed-article:8598453 | lifeskim:mentions | umls-concept:C1880389 | lld:lifeskim |
| pubmed-article:8598453 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:8598453 | pubmed:dateCreated | 1996-4-23 | lld:pubmed |
| pubmed-article:8598453 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8598453 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8598453 | pubmed:databankReference | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8598453 | pubmed:abstractText | FAS/Apo-1 (CD95) is an apoptosis-signaling cell surface receptor belonging to the TNF receptor family. Tumor cells resistant to Fas-mediated apoptosis have been described, but to date, the mechanisms responsible for this resistance are not well understood. We found that a series of apoptosis-resistant clones from human HUT78 lymphoma cells express a splicing variant coding for a truncated Fas molecule that lacks the intracellular death-signaling domain. The mutation responsible for the FasExo8Del expression was identified as a deletion-insertion in the intron 7/exon 8 region of the Fas gene. Moreover this mutation affects the phenotype in a dominant negative fashion, i.e., even in the presence of the normal receptor. | lld:pubmed |
| pubmed-article:8598453 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8598453 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8598453 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8598453 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:8598453 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:8598453 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8598453 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8598453 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:8598453 | pubmed:issn | 0022-1767 | lld:pubmed |
| pubmed-article:8598453 | pubmed:author | pubmed-author:TestaJJ | lld:pubmed |
| pubmed-article:8598453 | pubmed:author | pubmed-author:De MariaRR | lld:pubmed |
| pubmed-article:8598453 | pubmed:author | pubmed-author:CascinoII | lld:pubmed |
| pubmed-article:8598453 | pubmed:author | pubmed-author:RubertiGG | lld:pubmed |
| pubmed-article:8598453 | pubmed:author | pubmed-author:PapoffGG | lld:pubmed |
| pubmed-article:8598453 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8598453 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:8598453 | pubmed:volume | 156 | lld:pubmed |
| pubmed-article:8598453 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8598453 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8598453 | pubmed:pagination | 13-7 | lld:pubmed |
| pubmed-article:8598453 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:8598453 | pubmed:meshHeading | pubmed-meshheading:8598453-... | lld:pubmed |
| pubmed-article:8598453 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8598453 | pubmed:articleTitle | Fas/Apo-1 (CD95) receptor lacking the intracytoplasmic signaling domain protects tumor cells from Fas-mediated apoptosis. | lld:pubmed |
| pubmed-article:8598453 | pubmed:affiliation | Department of Immunobiology, National Research Council, Rome, Italy. | lld:pubmed |
| pubmed-article:8598453 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8598453 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8598453 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:355 | entrezgene:pubmed | pubmed-article:8598453 | lld:entrezgene |
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