| pubmed-article:8562970 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8562970 | lifeskim:mentions | umls-concept:C0086282 | lld:lifeskim |
| pubmed-article:8562970 | lifeskim:mentions | umls-concept:C0007589 | lld:lifeskim |
| pubmed-article:8562970 | lifeskim:mentions | umls-concept:C0007620 | lld:lifeskim |
| pubmed-article:8562970 | lifeskim:mentions | umls-concept:C0376515 | lld:lifeskim |
| pubmed-article:8562970 | lifeskim:mentions | umls-concept:C0439677 | lld:lifeskim |
| pubmed-article:8562970 | lifeskim:mentions | umls-concept:C0023636 | lld:lifeskim |
| pubmed-article:8562970 | lifeskim:mentions | umls-concept:C1511938 | lld:lifeskim |
| pubmed-article:8562970 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:8562970 | pubmed:dateCreated | 1996-3-1 | lld:pubmed |
| pubmed-article:8562970 | pubmed:abstractText | A1, a bcl-2 family member, has been identified as a hematopoietic-specific, early inducible gene. In this study it is shown that stable transfection of A1 into an interleukin-3 (IL-3)-dependent myeloid precursor cell line, 32D c13, leads to a retardation of IL-3 withdrawal-induced cell death similar to that observed with transfection of bcl-2. However, unlike bcl-2. A1 expression permits the accumulation of differentiated myeloid cells both before and after IL-3 withdrawal. Total cell accumulation, on the other hand, is considerably greater after IL-3 deprivation in the bcl-2 transfectant than in A1-expressing cells. Cells cotransfected with the two genes behave similarly to cells singly transfected with bcl-2, except that viability following IL-3 withdrawal is somewhat further enhanced. These results suggest that these two proteins have distinct roles that may be related to the divergent regulation of their expression during myeloid differentiation. | lld:pubmed |
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| pubmed-article:8562970 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8562970 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:8562970 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8562970 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:8562970 | pubmed:issn | 0006-4971 | lld:pubmed |
| pubmed-article:8562970 | pubmed:author | pubmed-author:ReedJ CJC | lld:pubmed |
| pubmed-article:8562970 | pubmed:author | pubmed-author:PrystowskyM... | lld:pubmed |
| pubmed-article:8562970 | pubmed:author | pubmed-author:MinD YDY | lld:pubmed |
| pubmed-article:8562970 | pubmed:author | pubmed-author:WangH GHG | lld:pubmed |
| pubmed-article:8562970 | pubmed:author | pubmed-author:OrlofskyAA | lld:pubmed |
| pubmed-article:8562970 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8562970 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:8562970 | pubmed:volume | 87 | lld:pubmed |
| pubmed-article:8562970 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8562970 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8562970 | pubmed:pagination | 983-92 | lld:pubmed |
| pubmed-article:8562970 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:8562970 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8562970 | pubmed:articleTitle | A1, a Bcl-2 family member, prolongs cell survival and permits myeloid differentiation. | lld:pubmed |
| pubmed-article:8562970 | pubmed:affiliation | Department of Pathology, Albert Einstein College of Medicine, Bronx, NY 10467, USA. | lld:pubmed |
| pubmed-article:8562970 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8562970 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:8562970 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8562970 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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