| pubmed-article:8536983 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8536983 | lifeskim:mentions | umls-concept:C1138842 | lld:lifeskim |
| pubmed-article:8536983 | lifeskim:mentions | umls-concept:C0029246 | lld:lifeskim |
| pubmed-article:8536983 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:8536983 | pubmed:dateCreated | 1996-2-8 | lld:pubmed |
| pubmed-article:8536983 | pubmed:abstractText | In heterozygotes, R-stippled (R-st) reduces the pigmenting potential of sensitive r alleles heritably (paramutation). R-st is comprised of four r genes arranged in direct orientation. Unequal crossing over within R-st generates deletion products retaining from one to three r genes. Paramutagenic strength decreased in parallel with copy number, both among internal and distal deletions. Single-gene R-st derivatives were nonparamutagenic. This was so whether or not the single gene retained the transposable element (I-R) responsible for seed spotting. Adding back r genes by intragenic recombination increased paramutagenicity in proportion to total gene number. Each member of a set of overlapping deletions retained moderately strong activity, showing that no single r gene or intragenic region is required for paramutagenicity. Proximal and distal loss R-st derivatives, each retaining two r genes, were less paramutagenic in trans than the corresponding four copy cis combination, indicating R-st's paramutagenic determinants function as a cis-interdependent unit in bringing about modification of a sensitive allele. | lld:pubmed |
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| pubmed-article:8536983 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8536983 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:8536983 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8536983 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:8536983 | pubmed:issn | 0016-6731 | lld:pubmed |
| pubmed-article:8536983 | pubmed:author | pubmed-author:AllemanMM | lld:pubmed |
| pubmed-article:8536983 | pubmed:author | pubmed-author:KermicleJ LJL | lld:pubmed |
| pubmed-article:8536983 | pubmed:author | pubmed-author:EgglestonW... | lld:pubmed |
| pubmed-article:8536983 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8536983 | pubmed:volume | 141 | lld:pubmed |
| pubmed-article:8536983 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8536983 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8536983 | pubmed:pagination | 361-72 | lld:pubmed |
| pubmed-article:8536983 | pubmed:dateRevised | 2010-9-13 | lld:pubmed |
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| pubmed-article:8536983 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:8536983 | pubmed:articleTitle | Organization of paramutagenicity in R-stippled maize. | lld:pubmed |
| pubmed-article:8536983 | pubmed:affiliation | Laboratory of Genetics, University of Wisconsin, Madison 53706, USA. | lld:pubmed |
| pubmed-article:8536983 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8536983 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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