| pubmed-article:8342595 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8342595 | lifeskim:mentions | umls-concept:C0021368 | lld:lifeskim |
| pubmed-article:8342595 | lifeskim:mentions | umls-concept:C0006104 | lld:lifeskim |
| pubmed-article:8342595 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:8342595 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:8342595 | lifeskim:mentions | umls-concept:C0014072 | lld:lifeskim |
| pubmed-article:8342595 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:8342595 | lifeskim:mentions | umls-concept:C0332120 | lld:lifeskim |
| pubmed-article:8342595 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:8342595 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:8342595 | pubmed:dateCreated | 1993-9-2 | lld:pubmed |
| pubmed-article:8342595 | pubmed:abstractText | In experimental autoimmune encephalomyelitis (EAE) myelin-specific T lymphocytes attack the myelinated tissue of the central nervous system (CNS). In the Lewis rat, EAE as a rule has an acute, monophasic course. With spontaneous clinical recovery the inflammatory CNS infiltrates are cleared from the nervous tissue within a few days. This is well in line with the remarkably low incidence of myelin-specific T cells present in EAE infiltrate. Combining immunocytochemical techniques, ultrastructural criteria and in situ nick translation we found up to 49% of T lymphocytes in EAE lesions showing signs of apoptosis at the time of recovery from disease. Our results suggest that apoptosis of T lymphocytes may be one possible mechanism to eliminate T lymphocytes from inflammatory brain lesions. | lld:pubmed |
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| pubmed-article:8342595 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8342595 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8342595 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:8342595 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8342595 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:8342595 | pubmed:issn | 0002-9440 | lld:pubmed |
| pubmed-article:8342595 | pubmed:author | pubmed-author:GoldRR | lld:pubmed |
| pubmed-article:8342595 | pubmed:author | pubmed-author:LassmannHH | lld:pubmed |
| pubmed-article:8342595 | pubmed:author | pubmed-author:WekerleHH | lld:pubmed |
| pubmed-article:8342595 | pubmed:author | pubmed-author:RotheGG | lld:pubmed |
| pubmed-article:8342595 | pubmed:author | pubmed-author:ZischlerHH | lld:pubmed |
| pubmed-article:8342595 | pubmed:author | pubmed-author:BreitschopfHH | lld:pubmed |
| pubmed-article:8342595 | pubmed:author | pubmed-author:SchmidtZZ | lld:pubmed |
| pubmed-article:8342595 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8342595 | pubmed:volume | 143 | lld:pubmed |
| pubmed-article:8342595 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8342595 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8342595 | pubmed:pagination | 446-52 | lld:pubmed |
| pubmed-article:8342595 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:8342595 | pubmed:year | 1993 | lld:pubmed |
| pubmed-article:8342595 | pubmed:articleTitle | Apoptosis of T lymphocytes in experimental autoimmune encephalomyelitis. Evidence for programmed cell death as a mechanism to control inflammation in the brain. | lld:pubmed |
| pubmed-article:8342595 | pubmed:affiliation | Research Unit of Experimental Neuropathology, Austrian Academy of Sciences, Vienna. | lld:pubmed |
| pubmed-article:8342595 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8342595 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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