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pubmed-article:8230281pubmed:abstractTextThe biotransformation of the natural estrogen 17 beta-estradiol (E2) via the C16 alpha-hydroxylation pathway is elevated in patients with breast cancer, in subjects at increased risk for developing breast cancer, and in c-Ha-ras-initiated mouse mammary epithelial cells.lld:pubmed
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pubmed-article:8230281pubmed:articleTitleUpregulation of estradiol C16 alpha-hydroxylation in human breast tissue: a potential biomarker of breast cancer risk.lld:pubmed
pubmed-article:8230281pubmed:affiliationStrang-Cornell Cancer Research Laboratory, Strand Cancer Prevention Center, New York, N.Y. 10021.lld:pubmed
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