pubmed-article:8165224 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:8165224 | lifeskim:mentions | umls-concept:C0003577 | lld:lifeskim |
pubmed-article:8165224 | lifeskim:mentions | umls-concept:C0039062 | lld:lifeskim |
pubmed-article:8165224 | lifeskim:mentions | umls-concept:C0007585 | lld:lifeskim |
pubmed-article:8165224 | lifeskim:mentions | umls-concept:C0206249 | lld:lifeskim |
pubmed-article:8165224 | lifeskim:mentions | umls-concept:C0598352 | lld:lifeskim |
pubmed-article:8165224 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
pubmed-article:8165224 | pubmed:issue | 1343 | lld:pubmed |
pubmed-article:8165224 | pubmed:dateCreated | 1994-5-20 | lld:pubmed |
pubmed-article:8165224 | pubmed:abstractText | Long-term potentiation (LTP) has been proposed as a cellular mechanism for associative learning in vertebrates. Induction of one type of LTP--observed at synapses in the CA1 region of the mammalian hippocampus--is regulated by the voltage of the postsynaptic cell. To date, a similar form of LTP has not been demonstrated for any invertebrate synapse. We now report that high-frequency stimulation can induce LTP of sensorimotor synapses of the marine mollusc Aplysia in cell culture. Moreover, induction of this form of LTP appears to involve a voltage-dependent postsynaptic mechanism because pairing tetanic stimulation of the presynaptic cell with strong hyperpolarization of the postsynaptic cell blocks the induction of LTP. | lld:pubmed |
pubmed-article:8165224 | pubmed:language | eng | lld:pubmed |
pubmed-article:8165224 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8165224 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:8165224 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:8165224 | pubmed:month | Feb | lld:pubmed |
pubmed-article:8165224 | pubmed:issn | 0962-8452 | lld:pubmed |
pubmed-article:8165224 | pubmed:author | pubmed-author:GlanzmanD LDL | lld:pubmed |
pubmed-article:8165224 | pubmed:author | pubmed-author:LinX YXY | lld:pubmed |
pubmed-article:8165224 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:8165224 | pubmed:day | 22 | lld:pubmed |
pubmed-article:8165224 | pubmed:volume | 255 | lld:pubmed |
pubmed-article:8165224 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:8165224 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:8165224 | pubmed:pagination | 113-8 | lld:pubmed |
pubmed-article:8165224 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
pubmed-article:8165224 | pubmed:meshHeading | pubmed-meshheading:8165224-... | lld:pubmed |
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pubmed-article:8165224 | pubmed:meshHeading | pubmed-meshheading:8165224-... | lld:pubmed |
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pubmed-article:8165224 | pubmed:meshHeading | pubmed-meshheading:8165224-... | lld:pubmed |
pubmed-article:8165224 | pubmed:year | 1994 | lld:pubmed |
pubmed-article:8165224 | pubmed:articleTitle | Long-term potentiation of Aplysia sensorimotor synapses in cell culture: regulation by postsynaptic voltage. | lld:pubmed |
pubmed-article:8165224 | pubmed:affiliation | Department of Physiological Science, University of California, Los Angeles 90024-1568. | lld:pubmed |
pubmed-article:8165224 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:8165224 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:8165224 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:8165224 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
pubmed-article:8165224 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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