| pubmed-article:8155283 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0025260 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0002708 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0023660 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0035397 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0392747 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0301630 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0599946 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C1704240 | lld:lifeskim |
| pubmed-article:8155283 | lifeskim:mentions | umls-concept:C0443172 | lld:lifeskim |
| pubmed-article:8155283 | pubmed:issue | 1-2 | lld:pubmed |
| pubmed-article:8155283 | pubmed:dateCreated | 1994-5-19 | lld:pubmed |
| pubmed-article:8155283 | pubmed:abstractText | The present study used a reward reduction paradigm to examine the role of the amygdala in memory for reduction in reward magnitude. Male Sprague-Dawley rats were implanted with bilateral amygdala cannulae and trained to run a straight alley (6 trials/day) for either ten or one 45-mg food pellets. On Day 10 of training, half the animals in the 10 pellet reward group were shifted to a one pellet reward. Immediately following shifted trials, the animals received an intra-amygdala injection of either a 2% lidocaine solution or phosphate buffer (0.5 microliter/side). Shifted training continued for 2 more days and no further injections were given. Shifted animals that received a buffer injection displayed a sharp increase in response latencies compared to unshifted controls on the second day of shifted training. In contrast, shifted animals that received intra-amygdala injections of lidocaine exhibited significantly lower latencies compared to the shifted vehicle group on the second day of shifted training. The findings indicate that post-training inactivation of the amygdala attenuates the response to reward reduction, suggesting that the amygdala modulates the storage for a reduction in reward magnitude. | lld:pubmed |
| pubmed-article:8155283 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8155283 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8155283 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8155283 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8155283 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:8155283 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8155283 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8155283 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:8155283 | pubmed:issn | 0166-4328 | lld:pubmed |
| pubmed-article:8155283 | pubmed:author | pubmed-author:McGaughJ LJL | lld:pubmed |
| pubmed-article:8155283 | pubmed:author | pubmed-author:PackardM GMG | lld:pubmed |
| pubmed-article:8155283 | pubmed:author | pubmed-author:SalinasJ AJA | lld:pubmed |
| pubmed-article:8155283 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8155283 | pubmed:day | 31 | lld:pubmed |
| pubmed-article:8155283 | pubmed:volume | 59 | lld:pubmed |
| pubmed-article:8155283 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8155283 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8155283 | pubmed:pagination | 153-9 | lld:pubmed |
| pubmed-article:8155283 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:meshHeading | pubmed-meshheading:8155283-... | lld:pubmed |
| pubmed-article:8155283 | pubmed:year | 1993 | lld:pubmed |
| pubmed-article:8155283 | pubmed:articleTitle | Amygdala modulates memory for changes in reward magnitude: reversible post-training inactivation with lidocaine attenuates the response to a reduction in reward. | lld:pubmed |
| pubmed-article:8155283 | pubmed:affiliation | Center for the Neurobiology of Learning and Memory, University of California at Irvine 92717-3800. | lld:pubmed |
| pubmed-article:8155283 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8155283 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8155283 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:8155283 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:8155283 | lld:pubmed |
