| pubmed-article:8151773 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8151773 | lifeskim:mentions | umls-concept:C0079337 | lld:lifeskim |
| pubmed-article:8151773 | lifeskim:mentions | umls-concept:C0042769 | lld:lifeskim |
| pubmed-article:8151773 | lifeskim:mentions | umls-concept:C0521026 | lld:lifeskim |
| pubmed-article:8151773 | lifeskim:mentions | umls-concept:C0178539 | lld:lifeskim |
| pubmed-article:8151773 | lifeskim:mentions | umls-concept:C1306673 | lld:lifeskim |
| pubmed-article:8151773 | lifeskim:mentions | umls-concept:C0205221 | lld:lifeskim |
| pubmed-article:8151773 | lifeskim:mentions | umls-concept:C0205225 | lld:lifeskim |
| pubmed-article:8151773 | lifeskim:mentions | umls-concept:C1521840 | lld:lifeskim |
| pubmed-article:8151773 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:8151773 | pubmed:dateCreated | 1994-5-12 | lld:pubmed |
| pubmed-article:8151773 | pubmed:abstractText | The objective of this study was to identify cellular and organ targets of acute feline immunodeficiency virus (FIV) infection in vivo. Tissues of FIV-infected cats were studied at eight time points during the first 3 months after experimental infection. FIV nucleic acids were first detected by in situ hybridization 21 days after infection, approximately 1.5 weeks after lymph node enlargement was first observed and 3 weeks before the primary acute flu-like illness. The majority of FIV-infected cells were present in lymphoid organs, though low numbers of infected cells were noted in nonlymphoid organs as well. Germinal centers harbored many of the FIV-infected cells within lymphoid tissues. The thymic cortex was also a major site of early infection. Combined in situ hybridization and immunohistochemistry revealed that T lymphocytes were the primary target of early FIV infection in tissues of cats before the onset of clinical signs of acute illness. An unidentified population of mononuclear cells and a few macrophages were also infected. During the ensuing acute flu-like illness, the proportion of FIV-infected macrophages in tissues increased dramatically. This early shift in the predominant cellular localization of FIV from T lymphocytes to macrophages may be important for establishing viral persistence. | lld:pubmed |
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| pubmed-article:8151773 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8151773 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8151773 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8151773 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8151773 | pubmed:month | May | lld:pubmed |
| pubmed-article:8151773 | pubmed:issn | 0022-538X | lld:pubmed |
| pubmed-article:8151773 | pubmed:author | pubmed-author:PedersenN CNC | lld:pubmed |
