pubmed-article:8053598 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:8053598 | lifeskim:mentions | umls-concept:C0085979 | lld:lifeskim |
pubmed-article:8053598 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:8053598 | lifeskim:mentions | umls-concept:C1302234 | lld:lifeskim |
pubmed-article:8053598 | lifeskim:mentions | umls-concept:C0241913 | lld:lifeskim |
pubmed-article:8053598 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:8053598 | pubmed:dateCreated | 1994-9-8 | lld:pubmed |
pubmed-article:8053598 | pubmed:abstractText | In the guinea pig, depleting hepatic glutathione before inhaling subanesthetic 0.1% halothane increases covalent binding of halothane biotransformation intermediates to hepatic protein and potentiates resultant liver injury. Because inhalation of a higher concentration of halothane is known to produce greater levels of covalent binding than with subanesthetic halothane, this study was undertaken with 0.25-1.0% halothane concentrations to further examine glutathione depletion as an etiology for halothane hepatitis. | lld:pubmed |
pubmed-article:8053598 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8053598 | pubmed:language | eng | lld:pubmed |
pubmed-article:8053598 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8053598 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:8053598 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8053598 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8053598 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8053598 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8053598 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:8053598 | pubmed:month | Aug | lld:pubmed |
pubmed-article:8053598 | pubmed:issn | 0003-3022 | lld:pubmed |
pubmed-article:8053598 | pubmed:author | pubmed-author:GandolfiA JAJ | lld:pubmed |
pubmed-article:8053598 | pubmed:author | pubmed-author:HallP MPM | lld:pubmed |
pubmed-article:8053598 | pubmed:author | pubmed-author:LinkR JRJ | lld:pubmed |
pubmed-article:8053598 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:8053598 | pubmed:volume | 81 | lld:pubmed |
pubmed-article:8053598 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:8053598 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:8053598 | pubmed:pagination | 478-87 | lld:pubmed |
pubmed-article:8053598 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
pubmed-article:8053598 | pubmed:meshHeading | pubmed-meshheading:8053598-... | lld:pubmed |
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pubmed-article:8053598 | pubmed:meshHeading | pubmed-meshheading:8053598-... | lld:pubmed |
pubmed-article:8053598 | pubmed:year | 1994 | lld:pubmed |
pubmed-article:8053598 | pubmed:articleTitle | A model for fatal halothane hepatitis in the guinea pig. | lld:pubmed |
pubmed-article:8053598 | pubmed:affiliation | Department of Anesthesiology, College of Medicine, University of Arizona, Tucson. | lld:pubmed |
pubmed-article:8053598 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:8053598 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |