| pubmed-article:8014908 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C2349001 | lld:lifeskim |
| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C0681850 | lld:lifeskim |
| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C1706203 | lld:lifeskim |
| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C2697811 | lld:lifeskim |
| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C0004388 | lld:lifeskim |
| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C1550501 | lld:lifeskim |
| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C0871269 | lld:lifeskim |
| pubmed-article:8014908 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:8014908 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:8014908 | pubmed:dateCreated | 1994-7-22 | lld:pubmed |
| pubmed-article:8014908 | pubmed:abstractText | 1. We studied eight young men (age range: 20-37 years) with chronic, clinically complete high cervical spinal cord injuries and ten age-matched healthy men to determine how interruption of connections between the central nervous system and spinal sympathetic motoneurones affects autonomic cardiovascular control. 2. Baseline diastolic pressures and R-R intervals (heart periods) were similar in the two groups. Slopes of R-R interval responses to brief neck pressure changes were significantly lower in tetraplegic than in healthy subjects, but slopes of R-R interval responses to steady-state arterial pressure reductions and increases were comparable. Plasma noradrenaline levels did not change significantly during steady-state arterial pressure reductions in tetraplegic patients, but rose sharply in healthy subjects. The range of arterial pressure and R-R interval responses to vasoactive drugs (nitroprusside and phenylephrine) was significantly greater in tetraplegic than healthy subjects. 3. Resting R-R interval spectral power at respiratory and low frequencies was similar in the two groups. During infusions of vasoactive drugs, low-frequency R-R interval spectral power was directly proportional to arterial pressure in tetraplegic patients, but was unrelated to arterial pressure in healthy subjects. Vagolytic doses of atropine nearly abolished both low- and respiratory-frequency R-R interval spectral power in both groups. 4. Our conclusions are as follows. First, since tetraplegic patients have significant levels of low-frequency arterial pressure and R-R interval spectral power, human Mayer arterial pressure waves may result from mechanisms that do not involve stimulation of spinal sympathetic motoneurones by brainstem neurones. Second, since in tetraplegic patients, low-frequency R-R interval spectral power is proportional to arterial pressure, it is likely to be mediated by a baroreflex mechanism. Third, since low-frequency R-R interval rhythms were nearly abolished by atropine in both tetraplegic and healthy subjects, these rhythms reflect in an important way rhythmic firing of vagal cardiac motoneurones. | lld:pubmed |
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| pubmed-article:8014908 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:8014908 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:8014908 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8014908 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:8014908 | pubmed:issn | 0022-3751 | lld:pubmed |
| pubmed-article:8014908 | pubmed:author | pubmed-author:EckbergD LDL | lld:pubmed |
| pubmed-article:8014908 | pubmed:author | pubmed-author:BrownT ETE | lld:pubmed |
| pubmed-article:8014908 | pubmed:author | pubmed-author:KooEE | lld:pubmed |
| pubmed-article:8014908 | pubmed:author | pubmed-author:BeightolL ALA | lld:pubmed |
| pubmed-article:8014908 | pubmed:author | pubmed-author:GuC HCH | lld:pubmed |
| pubmed-article:8014908 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8014908 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:8014908 | pubmed:volume | 474 | lld:pubmed |
| pubmed-article:8014908 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8014908 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8014908 | pubmed:pagination | 483-95 | lld:pubmed |
| pubmed-article:8014908 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:8014908 | pubmed:year | 1994 | lld:pubmed |
| pubmed-article:8014908 | pubmed:articleTitle | Human autonomic rhythms: vagal cardiac mechanisms in tetraplegic subjects. | lld:pubmed |
| pubmed-article:8014908 | pubmed:affiliation | Department of Medicine, Hunter Holmes McGuire Department of Veterans Affairs Medical Center, Richmond, VA. | lld:pubmed |
| pubmed-article:8014908 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8014908 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8014908 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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