pubmed-article:8010857 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:8010857 | lifeskim:mentions | umls-concept:C0039070 | lld:lifeskim |
pubmed-article:8010857 | lifeskim:mentions | umls-concept:C0007194 | lld:lifeskim |
pubmed-article:8010857 | lifeskim:mentions | umls-concept:C0235480 | lld:lifeskim |
pubmed-article:8010857 | lifeskim:mentions | umls-concept:C1524003 | lld:lifeskim |
pubmed-article:8010857 | lifeskim:mentions | umls-concept:C1542147 | lld:lifeskim |
pubmed-article:8010857 | pubmed:issue | 11 | lld:pubmed |
pubmed-article:8010857 | pubmed:dateCreated | 1994-7-18 | lld:pubmed |
pubmed-article:8010857 | pubmed:abstractText | The aim of this retrospective study was to determine the mechanism of syncope in idiopathic hypertrophic cardiomyopathy (HCM). An electrocardiographic study was undertaken in 43 patients with HCM: 27 (Group I) had a history of syncope and 16 (Group II) had no history of syncope but were investigated for conduction defects (n = 7) or unsustained ventricular tachycardia (VT) (n = 9). The stimulation protocol used programmed atrial pacing with 1 and 2 extrastimuli and ventricular pacing using up to 3 extrastimuli delivered at 2 sites. The following results were obtained: sustained atrial fibrillation (AF) (> 1 min) was induced in 21 patients in Group I (78%), 4 in Group II (25%); VT was induced in 3 patients in Group I (11%), and 3 in Group II (19%); infra-Hisian block was detected in 1 patient in Group I. The mechanism of syncope was elucidated in 23 patients in Group I (85%): one atrioventricular block 1 sinus node dysfunction, 18 atrial fibrillations, 2 associations of AF-VT and 1 VT. The authors conclude that the prevalence of inducible AF was higher in patients with HCA and syncope than in controls and HCM without syncope: this was the only detectable difference in 67% of patients with unexplained syncope. Paroxysmal AF could therefore explain malaise or syncope in up to 2/3 of cases of HCM. | lld:pubmed |
pubmed-article:8010857 | pubmed:language | fre | lld:pubmed |
pubmed-article:8010857 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8010857 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:8010857 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8010857 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:8010857 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:8010857 | pubmed:month | Nov | lld:pubmed |
pubmed-article:8010857 | pubmed:issn | 0003-9683 | lld:pubmed |
pubmed-article:8010857 | pubmed:author | pubmed-author:BeurrierDD | lld:pubmed |
pubmed-article:8010857 | pubmed:author | pubmed-author:Brembilla-Per... | lld:pubmed |
pubmed-article:8010857 | pubmed:author | pubmed-author:Terrier de... | lld:pubmed |
pubmed-article:8010857 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:8010857 | pubmed:volume | 86 | lld:pubmed |
pubmed-article:8010857 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:8010857 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:8010857 | pubmed:pagination | 1573-8 | lld:pubmed |
pubmed-article:8010857 | pubmed:dateRevised | 2009-2-13 | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:meshHeading | pubmed-meshheading:8010857-... | lld:pubmed |
pubmed-article:8010857 | pubmed:year | 1993 | lld:pubmed |
pubmed-article:8010857 | pubmed:articleTitle | [Paroxysmal atrial fibrillation: main cause of syncope in hypertrophic cardiomyopathy]. | lld:pubmed |
pubmed-article:8010857 | pubmed:affiliation | Service de cardiologie A, CHU Brabois, Vandoeuvre. | lld:pubmed |
pubmed-article:8010857 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:8010857 | pubmed:publicationType | English Abstract | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:8010857 | lld:pubmed |