| pubmed-article:7960100 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7960100 | lifeskim:mentions | umls-concept:C0031921 | lld:lifeskim |
| pubmed-article:7960100 | lifeskim:mentions | umls-concept:C0085478 | lld:lifeskim |
| pubmed-article:7960100 | lifeskim:mentions | umls-concept:C0024432 | lld:lifeskim |
| pubmed-article:7960100 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
| pubmed-article:7960100 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:7960100 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:7960100 | pubmed:dateCreated | 1994-12-29 | lld:pubmed |
| pubmed-article:7960100 | pubmed:abstractText | The present study was performed to examine whether Porphyromonas gingivalis fimbriae induce specifically a protein kinase-mediated phosphorylated protein that is involved in the mechanism of signal transduction. The fimbriae induced a 68-kDa phosphorylated protein (pp68) in a dose-dependent manner in mouse peritoneal macrophages. A marked appearance of pp68 was observed 20 min after the initiation of fimbrial treatment. The fimbria-induced pp68 was inhibited dramatically by staurosporine, a potent inhibitor of protein kinase C. pp68 induction was also inhibited by H-7, a potent inhibitor of several types of protein kinase. However, the induction was not inhibited by HA-1004 and H-8, relatively high-affinity inhibitors of protein kinase A. Phorbol myristate acetate and 1-oleoyl-2-acetyl-sn-glycerol, activators of protein kinase C, were able to induce pp68 in mouse peritoneal macrophages. This protein was localized in the cytosolic fraction of fimbria-treated macrophages. pp68 also was induced in fimbria-treated human monocyte-like cells. Finally, we observed that gene expression of the fimbria-induced neutrophil chemoattractant KC was inhibited markedly by staurosporine. | lld:pubmed |
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| pubmed-article:7960100 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7960100 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7960100 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7960100 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7960100 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:7960100 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:7960100 | pubmed:author | pubmed-author:WatanabeAA | lld:pubmed |
| pubmed-article:7960100 | pubmed:author | pubmed-author:KitanoSS | lld:pubmed |
| pubmed-article:7960100 | pubmed:author | pubmed-author:MurakamiYY | lld:pubmed |
| pubmed-article:7960100 | pubmed:author | pubmed-author:KawakamiKK | lld:pubmed |
| pubmed-article:7960100 | pubmed:author | pubmed-author:IwasakaHH | lld:pubmed |
| pubmed-article:7960100 | pubmed:author | pubmed-author:HanazawaSS | lld:pubmed |
| pubmed-article:7960100 | pubmed:author | pubmed-author:NaganumaKK | lld:pubmed |
| pubmed-article:7960100 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7960100 | pubmed:volume | 62 | lld:pubmed |
| pubmed-article:7960100 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7960100 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7960100 | pubmed:pagination | 5242-6 | lld:pubmed |
| pubmed-article:7960100 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:7960100 | pubmed:year | 1994 | lld:pubmed |
| pubmed-article:7960100 | pubmed:articleTitle | Porphyromonas gingivalis fimbriae induce a 68-kilodalton phosphorylated protein in macrophages. | lld:pubmed |
| pubmed-article:7960100 | pubmed:affiliation | Department of Oral Microbiology, Meikai University School of Dentistry, Saitama, Japan. | lld:pubmed |
| pubmed-article:7960100 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7960100 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:7960100 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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