| pubmed-article:7906942 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7906942 | lifeskim:mentions | umls-concept:C0007600 | lld:lifeskim |
| pubmed-article:7906942 | lifeskim:mentions | umls-concept:C0123759 | lld:lifeskim |
| pubmed-article:7906942 | lifeskim:mentions | umls-concept:C0024432 | lld:lifeskim |
| pubmed-article:7906942 | lifeskim:mentions | umls-concept:C1515670 | lld:lifeskim |
| pubmed-article:7906942 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:7906942 | lifeskim:mentions | umls-concept:C0591833 | lld:lifeskim |
| pubmed-article:7906942 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:7906942 | pubmed:dateCreated | 1994-3-29 | lld:pubmed |
| pubmed-article:7906942 | pubmed:abstractText | Interleukin (IL)-12 is a logical candidate for participation in the differentiation of T helper (Th) 1 cells. IL-12 is produced by macrophages and B cells and induces the production of interferon (IFN)-gamma from Th1 cells and NK cells. In this study, we show that the IFN-gamma itself is capable of inducing IL-12 mRNA expression in murine macrophage cell line J774. The mRNA was apparent by 12h after IFN-gamma treatment, although maximal induction required 36 to 48h. Furthermore, we investigated the signal transduction mechanism responsible for the mRNA expression. The IFN-gamma-inducible IL-12 mRNA was blocked by two protein tyrosine kinase (PTK) inhibitors, genistein and herbimycin A, suggesting that PTK is involved in the IFN-gamma-inducible IL-12 mRNA expression. | lld:pubmed |
| pubmed-article:7906942 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7906942 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7906942 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7906942 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7906942 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:7906942 | pubmed:issn | 0006-291X | lld:pubmed |
| pubmed-article:7906942 | pubmed:author | pubmed-author:YoshidaAA | lld:pubmed |
| pubmed-article:7906942 | pubmed:author | pubmed-author:KoideYY | lld:pubmed |
| pubmed-article:7906942 | pubmed:author | pubmed-author:YoshidaT OTO | lld:pubmed |
| pubmed-article:7906942 | pubmed:author | pubmed-author:UchijimaMM | lld:pubmed |
| pubmed-article:7906942 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7906942 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:7906942 | pubmed:volume | 198 | lld:pubmed |
| pubmed-article:7906942 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7906942 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7906942 | pubmed:pagination | 857-61 | lld:pubmed |
| pubmed-article:7906942 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:7906942 | pubmed:year | 1994 | lld:pubmed |
| pubmed-article:7906942 | pubmed:articleTitle | IFN-gamma induces IL-12 mRNA expression by a murine macrophage cell line, J774. | lld:pubmed |
| pubmed-article:7906942 | pubmed:affiliation | Department of Microbiology and Immunology, Hamamatsu University School of Medicine, Japan. | lld:pubmed |
| pubmed-article:7906942 | pubmed:publicationType | Journal Article | lld:pubmed |
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