| pubmed-article:7888258 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7888258 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:7888258 | lifeskim:mentions | umls-concept:C0026267 | lld:lifeskim |
| pubmed-article:7888258 | lifeskim:mentions | umls-concept:C0085612 | lld:lifeskim |
| pubmed-article:7888258 | lifeskim:mentions | umls-concept:C0332197 | lld:lifeskim |
| pubmed-article:7888258 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:7888258 | lifeskim:mentions | umls-concept:C0332624 | lld:lifeskim |
| pubmed-article:7888258 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:7888258 | pubmed:dateCreated | 1995-4-17 | lld:pubmed |
| pubmed-article:7888258 | pubmed:abstractText | The mechanism responsible for the reported high incidence of ventricular arrhythmias in mitral valve prolapse is not clear. Electrocardiographic studies show an increased occurrence of repolarisation abnormalities on the 12 lead surface electrocardiogram, indicating regional differences in ventricular recovery. The purpose of this study was to investigate whether dispersion of refractoriness was an arrhythmogenic mechanism. | lld:pubmed |
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| pubmed-article:7888258 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7888258 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7888258 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:7888258 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7888258 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:7888258 | pubmed:issn | 0007-0769 | lld:pubmed |
| pubmed-article:7888258 | pubmed:author | pubmed-author:LimK AKA | lld:pubmed |
| pubmed-article:7888258 | pubmed:author | pubmed-author:CrijnsH JHJ | lld:pubmed |
| pubmed-article:7888258 | pubmed:author | pubmed-author:WiesfeldA CAC | lld:pubmed |
| pubmed-article:7888258 | pubmed:author | pubmed-author:HawesR CRC | lld:pubmed |
| pubmed-article:7888258 | pubmed:author | pubmed-author:TielemanR GRG | lld:pubmed |
| pubmed-article:7888258 | pubmed:author | pubmed-author:PosmaJJ | lld:pubmed |
| pubmed-article:7888258 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7888258 | pubmed:volume | 73 | lld:pubmed |
| pubmed-article:7888258 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7888258 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7888258 | pubmed:pagination | 37-40 | lld:pubmed |
| pubmed-article:7888258 | pubmed:dateRevised | 2010-3-24 | lld:pubmed |
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| pubmed-article:7888258 | pubmed:meshHeading | pubmed-meshheading:7888258-... | lld:pubmed |
| pubmed-article:7888258 | pubmed:meshHeading | pubmed-meshheading:7888258-... | lld:pubmed |
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| pubmed-article:7888258 | pubmed:meshHeading | pubmed-meshheading:7888258-... | lld:pubmed |
| pubmed-article:7888258 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7888258 | pubmed:articleTitle | Increased dispersion of refractoriness in the absence of QT prolongation in patients with mitral valve prolapse and ventricular arrhythmias. | lld:pubmed |
| pubmed-article:7888258 | pubmed:affiliation | Department of Cardiology, University Hospital, Groningen, The Netherlands. | lld:pubmed |
| pubmed-article:7888258 | pubmed:publicationType | Journal Article | lld:pubmed |
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