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pubmed-article:7870264pubmed:abstractTextThe authors discuss the role of active neuropeptides Calcitonin Gene-Related Peptide (CGRP) and Vasoactive Intestinal Polypeptide (VIP) in the arterial spasm caused by haemorrhage from ruptured intracranial aneurysm. These substances act as vasodilators on brain vessels. Between the first and the second week after subarachnoid haemorrhage their concentration in the periarterial nerve fibres of the big vessels is significantly low. According to the experiments, the erythrocytes are this component of the blood which causes the decrease of the number of nerve fibres containing CGRP and VIP. The clinical tests of CGRP and VIP in the treatment of the vascular spasm after subarachnoid haemorrhage are being continued.lld:pubmed
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pubmed-article:7870264pubmed:authorpubmed-author:ZderkiewiczEElld:pubmed
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pubmed-article:7870264pubmed:authorpubmed-author:MosiewiczAAlld:pubmed
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pubmed-article:7870264pubmed:volume28lld:pubmed
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pubmed-article:7870264pubmed:pagination899-905lld:pubmed
pubmed-article:7870264pubmed:dateRevised2006-11-15lld:pubmed
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pubmed-article:7870264pubmed:articleTitle[Vasoactive neuropeptides CGRP (calcitonin gene-related peptide) and vasospasm during subarachnoid hemorrhage from ruptured intracranial aneurysm].lld:pubmed
pubmed-article:7870264pubmed:affiliationKatedry i Kliniki Neurochirurgii Akademii Medycznej, Lublinie.lld:pubmed
pubmed-article:7870264pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:7870264pubmed:publicationTypeEnglish Abstractlld:pubmed
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