| pubmed-article:7853958 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7853958 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:7853958 | lifeskim:mentions | umls-concept:C0012472 | lld:lifeskim |
| pubmed-article:7853958 | lifeskim:mentions | umls-concept:C0205227 | lld:lifeskim |
| pubmed-article:7853958 | pubmed:issue | 8947 | lld:pubmed |
| pubmed-article:7853958 | pubmed:dateCreated | 1995-3-10 | lld:pubmed |
| pubmed-article:7853958 | pubmed:abstractText | The possibility that impaired production of bronchoprotective factors contributes to the pathogenesis of asthma cannot be excluded. Prostaglandin E2 (PGE2) could be such a factor. It is a dominant cyclo-oxygenase product of airway epithelium and smooth muscle; it has inhibitory effects on inflammatory cells and pathways involved in bronchoconstriction at concentrations known to occur in the airway; inhalation of PGE2 has considerable bronchoprotective effects in patients with asthma; and manoeuvres that increase or decrease endogenous production of PGE2 have beneficial and deleterious effects on airway function. | lld:pubmed |
| pubmed-article:7853958 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7853958 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7853958 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7853958 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:7853958 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7853958 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7853958 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:7853958 | pubmed:issn | 0140-6736 | lld:pubmed |
| pubmed-article:7853958 | pubmed:author | pubmed-author:TattersfieldA... | lld:pubmed |
| pubmed-article:7853958 | pubmed:author | pubmed-author:PavordI DID | lld:pubmed |
| pubmed-article:7853958 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7853958 | pubmed:day | 18 | lld:pubmed |
| pubmed-article:7853958 | pubmed:volume | 345 | lld:pubmed |
| pubmed-article:7853958 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7853958 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7853958 | pubmed:pagination | 436-8 | lld:pubmed |
| pubmed-article:7853958 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:7853958 | pubmed:meshHeading | pubmed-meshheading:7853958-... | lld:pubmed |
| pubmed-article:7853958 | pubmed:meshHeading | pubmed-meshheading:7853958-... | lld:pubmed |
| pubmed-article:7853958 | pubmed:meshHeading | pubmed-meshheading:7853958-... | lld:pubmed |
| pubmed-article:7853958 | pubmed:meshHeading | pubmed-meshheading:7853958-... | lld:pubmed |
| pubmed-article:7853958 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7853958 | pubmed:articleTitle | Bronchoprotective role for endogenous prostaglandin E2. | lld:pubmed |
| pubmed-article:7853958 | pubmed:affiliation | Division of Respiratory Medicine, Nottingham University, City Hospital, UK. | lld:pubmed |
| pubmed-article:7853958 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7853958 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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