| pubmed-article:7783751 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7783751 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:7783751 | lifeskim:mentions | umls-concept:C0021017 | lld:lifeskim |
| pubmed-article:7783751 | lifeskim:mentions | umls-concept:C0206071 | lld:lifeskim |
| pubmed-article:7783751 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:7783751 | pubmed:dateCreated | 1995-7-17 | lld:pubmed |
| pubmed-article:7783751 | pubmed:abstractText | Mice with the x-linked immunodeficiency mutation (xid) are unresponsive to polysaccharide antigens, lack a subset of B cells, and have low serum IgM (2-20% of normal) and IgG3 (3% of normal). Because of the disproportionate reduction of IgG3, the ability of B cells from xid mice to switch to gamma 3 was examined. Switching was indirectly measured by comparing IgG3 production and C gamma 3 mRNA steady state levels of purified B cells activated to switch to IgG3 by LPS in bulk culture. Direct measurement of switching was achieved by enumerating on a percentage basis switched cells in a filter disk culture assay and by FACS analysis. In both bulk culture and the filter disk assay, switching to gamma 3 was equivalent between xid and non-xid B cells. | lld:pubmed |
| pubmed-article:7783751 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7783751 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783751 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:7783751 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783751 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783751 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783751 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7783751 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7783751 | pubmed:month | May | lld:pubmed |
| pubmed-article:7783751 | pubmed:issn | 0161-5890 | lld:pubmed |
| pubmed-article:7783751 | pubmed:author | pubmed-author:SteinK EKE | lld:pubmed |
| pubmed-article:7783751 | pubmed:author | pubmed-author:BrorsonK AKA | lld:pubmed |
| pubmed-article:7783751 | pubmed:author | pubmed-author:KrasnokutskyM... | lld:pubmed |
| pubmed-article:7783751 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7783751 | pubmed:volume | 32 | lld:pubmed |
| pubmed-article:7783751 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7783751 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7783751 | pubmed:pagination | 487-94 | lld:pubmed |
| pubmed-article:7783751 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:meshHeading | pubmed-meshheading:7783751-... | lld:pubmed |
| pubmed-article:7783751 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7783751 | pubmed:articleTitle | Immunoglobulin isotype switching in xid mice. | lld:pubmed |
| pubmed-article:7783751 | pubmed:affiliation | Division of Monoclonal Antibodies, Food and Drug Administration, Bethesda, MD 20892, USA. | lld:pubmed |
| pubmed-article:7783751 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7783751 | pubmed:publicationType | In Vitro | lld:pubmed |
| entrez-gene:12229 | entrezgene:pubmed | pubmed-article:7783751 | lld:entrezgene |
