| pubmed-article:7729674 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7729674 | lifeskim:mentions | umls-concept:C0038410 | lld:lifeskim |
| pubmed-article:7729674 | lifeskim:mentions | umls-concept:C0017398 | lld:lifeskim |
| pubmed-article:7729674 | lifeskim:mentions | umls-concept:C0441889 | lld:lifeskim |
| pubmed-article:7729674 | lifeskim:mentions | umls-concept:C0370215 | lld:lifeskim |
| pubmed-article:7729674 | lifeskim:mentions | umls-concept:C0030834 | lld:lifeskim |
| pubmed-article:7729674 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
| pubmed-article:7729674 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:7729674 | pubmed:dateCreated | 1995-5-31 | lld:pubmed |
| pubmed-article:7729674 | pubmed:abstractText | Mosaic penicillin-binding proteins (PBP) 1A, 2X and 2B genes were cloned from four clinical isolates of Streptococcus pneumoniae with levels of susceptibility to penicillin ranging from 1.5 to 16 micrograms benzylpenicillin ml-1. In each instance it was possible to transform either the penicillin-sensitive laboratory strain R6 or a sensitive clinical isolate 110K/70 to the full level of penicillin resistance with these three penicillin-binding proteins alone. Until now it has not been possible to clearly determine whether alterations to PBP1A, 2X and 2B alone were sufficient to attain high level penicillin resistance. | lld:pubmed |
| pubmed-article:7729674 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7729674 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7729674 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7729674 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7729674 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:7729674 | pubmed:issn | 0378-1097 | lld:pubmed |
| pubmed-article:7729674 | pubmed:author | pubmed-author:YenLL | lld:pubmed |
| pubmed-article:7729674 | pubmed:author | pubmed-author:DowsonC GCG | lld:pubmed |
| pubmed-article:7729674 | pubmed:author | pubmed-author:CoffeyT JTJ | lld:pubmed |
| pubmed-article:7729674 | pubmed:author | pubmed-author:BarcusV AVA | lld:pubmed |
| pubmed-article:7729674 | pubmed:author | pubmed-author:GhanekarKK | lld:pubmed |
| pubmed-article:7729674 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7729674 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:7729674 | pubmed:volume | 126 | lld:pubmed |
| pubmed-article:7729674 | pubmed:geneSymbol | PBP2X | lld:pubmed |
| pubmed-article:7729674 | pubmed:geneSymbol | PBP1A | lld:pubmed |
| pubmed-article:7729674 | pubmed:geneSymbol | PBP2B | lld:pubmed |
| pubmed-article:7729674 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7729674 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7729674 | pubmed:pagination | 299-303 | lld:pubmed |
| pubmed-article:7729674 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:7729674 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7729674 | pubmed:articleTitle | Genetics of high level penicillin resistance in clinical isolates of Streptococcus pneumoniae. | lld:pubmed |
| pubmed-article:7729674 | pubmed:affiliation | Molecular Microbiology Group, School of Biological Sciences, University of Sussex, Brighton, UK. | lld:pubmed |
| pubmed-article:7729674 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7729674 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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