| pubmed-article:7700015 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7700015 | lifeskim:mentions | umls-concept:C0001430 | lld:lifeskim |
| pubmed-article:7700015 | lifeskim:mentions | umls-concept:C0035094 | lld:lifeskim |
| pubmed-article:7700015 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:7700015 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:7700015 | pubmed:dateCreated | 1995-5-3 | lld:pubmed |
| pubmed-article:7700015 | pubmed:abstractText | Approximately one half of the aldosterone-producing adenomas (APA) removed from patients with primary aldosteronism in the Hypertension Unit at Greenslopes Hospital belong to a subgroup in which aldosterone levels are responsive to the renin-angiotensin system (angiotensin-responsive APA; AII-R-APA), unlike classical APAs in which aldosterone is unresponsive (AII-U-APA). Renin mRNA levels in AII-R-APA were elevated when compared with those in AII-U-APA or normal adrenal cortices. Renin mRNA levels in some adrenal cortices surrounding AII-R-APA (but never in AII-U-APA) were raised, suggesting that a genetic defect is not confined to the tumor. Renin gene RFLP analysis in peripheral blood DNA revealed a significant difference in allelic frequencies between patients with AII-R-APA and AII-U-APA, suggesting an association between an alteration in the renin gene and aldosterone responsiveness to the renin-angiotensin system in patients with APAs. | lld:pubmed |
| pubmed-article:7700015 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7700015 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7700015 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:7700015 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7700015 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7700015 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7700015 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7700015 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:7700015 | pubmed:issn | 0085-2538 | lld:pubmed |
| pubmed-article:7700015 | pubmed:author | pubmed-author:GordonR DRD | lld:pubmed |
| pubmed-article:7700015 | pubmed:author | pubmed-author:TunnyT JTJ | lld:pubmed |
| pubmed-article:7700015 | pubmed:author | pubmed-author:BallantineD... | lld:pubmed |
| pubmed-article:7700015 | pubmed:author | pubmed-author:StowasserMM | lld:pubmed |
| pubmed-article:7700015 | pubmed:author | pubmed-author:KlemmS ASA | lld:pubmed |
| pubmed-article:7700015 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7700015 | pubmed:volume | 46 | lld:pubmed |
| pubmed-article:7700015 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7700015 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7700015 | pubmed:pagination | 1591-3 | lld:pubmed |
| pubmed-article:7700015 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:meshHeading | pubmed-meshheading:7700015-... | lld:pubmed |
| pubmed-article:7700015 | pubmed:year | 1994 | lld:pubmed |
| pubmed-article:7700015 | pubmed:articleTitle | The renin gene and aldosterone-producing adenomas. | lld:pubmed |
| pubmed-article:7700015 | pubmed:affiliation | Hypertension Unit, Greenslopes Hospital, Brisbane, Australia. | lld:pubmed |
| pubmed-article:7700015 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7700015 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
