pubmed-article:7585807 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C1882687 | lld:lifeskim |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C0035696 | lld:lifeskim |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C0011501 | lld:lifeskim |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C0021655 | lld:lifeskim |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C0205216 | lld:lifeskim |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C0243144 | lld:lifeskim |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C1550605 | lld:lifeskim |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C0456205 | lld:lifeskim |
pubmed-article:7585807 | lifeskim:mentions | umls-concept:C0857121 | lld:lifeskim |
pubmed-article:7585807 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:7585807 | pubmed:dateCreated | 1995-12-12 | lld:pubmed |
pubmed-article:7585807 | pubmed:abstractText | Insulin resistance in skeletal muscle and adipose tissue often accompanies hypertension; however, it has not been shown that heart muscle is similarly affected. The aims of this study were to determine whether basal and insulin-stimulated glucose transport and glucose transporter mRNA content are altered in the spontaneously hypertensive rat (SHR) heart. | lld:pubmed |
pubmed-article:7585807 | pubmed:language | eng | lld:pubmed |
pubmed-article:7585807 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:7585807 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:7585807 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:7585807 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:7585807 | pubmed:month | Aug | lld:pubmed |
pubmed-article:7585807 | pubmed:issn | 0008-6363 | lld:pubmed |
pubmed-article:7585807 | pubmed:author | pubmed-author:RaddaG KGK | lld:pubmed |
pubmed-article:7585807 | pubmed:author | pubmed-author:HeathJJ | lld:pubmed |
pubmed-article:7585807 | pubmed:author | pubmed-author:PaternostroGG | lld:pubmed |
pubmed-article:7585807 | pubmed:author | pubmed-author:ClarkeKK | lld:pubmed |
pubmed-article:7585807 | pubmed:author | pubmed-author:SeymourA MAM | lld:pubmed |
pubmed-article:7585807 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:7585807 | pubmed:volume | 30 | lld:pubmed |
pubmed-article:7585807 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:7585807 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:7585807 | pubmed:pagination | 205-11 | lld:pubmed |
pubmed-article:7585807 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:7585807 | pubmed:year | 1995 | lld:pubmed |
pubmed-article:7585807 | pubmed:articleTitle | Decreased GLUT-4 mRNA content and insulin-sensitive deoxyglucose uptake show insulin resistance in the hypertensive rat heart. | lld:pubmed |
pubmed-article:7585807 | pubmed:affiliation | Department of Biochemistry, University of Oxford, UK. | lld:pubmed |
pubmed-article:7585807 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:7585807 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:7585807 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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