| pubmed-article:7544752 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7544752 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:7544752 | lifeskim:mentions | umls-concept:C0034721 | lld:lifeskim |
| pubmed-article:7544752 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:7544752 | lifeskim:mentions | umls-concept:C0001339 | lld:lifeskim |
| pubmed-article:7544752 | lifeskim:mentions | umls-concept:C0017710 | lld:lifeskim |
| pubmed-article:7544752 | lifeskim:mentions | umls-concept:C0086272 | lld:lifeskim |
| pubmed-article:7544752 | lifeskim:mentions | umls-concept:C0205227 | lld:lifeskim |
| pubmed-article:7544752 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:7544752 | pubmed:dateCreated | 1995-10-2 | lld:pubmed |
| pubmed-article:7544752 | pubmed:abstractText | Cytokines activate the hypothalamic-pituitary-adrenal axis and suppress inflammation by stimulating glucocorticoid secretion. The state of adrenocortical function during acute pancreatitis and its role in this disease were determined. | lld:pubmed |
| pubmed-article:7544752 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7544752 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7544752 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:7544752 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7544752 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:7544752 | pubmed:issn | 0016-5085 | lld:pubmed |
| pubmed-article:7544752 | pubmed:author | pubmed-author:AbeTT | lld:pubmed |
| pubmed-article:7544752 | pubmed:author | pubmed-author:KimuraKK | lld:pubmed |
| pubmed-article:7544752 | pubmed:author | pubmed-author:AbeRR | lld:pubmed |
| pubmed-article:7544752 | pubmed:author | pubmed-author:ToyotaTT | lld:pubmed |
| pubmed-article:7544752 | pubmed:author | pubmed-author:KoizumiMM | lld:pubmed |
| pubmed-article:7544752 | pubmed:author | pubmed-author:ShimosegawaTT | lld:pubmed |
| pubmed-article:7544752 | pubmed:author | pubmed-author:KashimuraJJ | lld:pubmed |
| pubmed-article:7544752 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7544752 | pubmed:volume | 109 | lld:pubmed |
| pubmed-article:7544752 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7544752 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7544752 | pubmed:pagination | 933-43 | lld:pubmed |
| pubmed-article:7544752 | pubmed:dateRevised | 2003-11-14 | lld:pubmed |
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| pubmed-article:7544752 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7544752 | pubmed:articleTitle | The role of endogenous glucocorticoids in rat experimental models of acute pancreatitis. | lld:pubmed |
| pubmed-article:7544752 | pubmed:affiliation | Third Department of Internal Medicine, Tohoku University School of Medicine, Miyagi, Japan. | lld:pubmed |
| pubmed-article:7544752 | pubmed:publicationType | Journal Article | lld:pubmed |
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