| pubmed-article:7542548 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C0567416 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C0004561 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C0007589 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C1514559 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C0037083 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C1413206 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C0205161 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C1527148 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:7542548 | lifeskim:mentions | umls-concept:C1511938 | lld:lifeskim |
| pubmed-article:7542548 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:7542548 | pubmed:dateCreated | 1995-8-30 | lld:pubmed |
| pubmed-article:7542548 | pubmed:abstractText | CD19-deficient mice were generated to examine the role of CD19 in B cell growth regulation in vivo. Deletion of CD19 had no deleterious effects on the generation of B cells in the bone marrow, but there was a significant reduction in the number of B cells in peripheral lymphoid tissues. B cells from CD19-deficient mice exhibited markedly decreased proliferative responses to mitogens, and serum immunoglobulin levels were also significantly decreased. In contrast, mice that overexpressed CD19 had significant defects in early B cell development in the bone marrow, augmented mitogenic responses, and increased serum immunoglobulin levels. These experiments indicate that CD19 functions to define signaling thresholds for cell surface receptors that regulate B lymphocyte selection, activation, and differentiation. | lld:pubmed |
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| pubmed-article:7542548 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7542548 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7542548 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7542548 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7542548 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:7542548 | pubmed:issn | 1074-7613 | lld:pubmed |
| pubmed-article:7542548 | pubmed:author | pubmed-author:SatoSS | lld:pubmed |
| pubmed-article:7542548 | pubmed:author | pubmed-author:EngelPP | lld:pubmed |
| pubmed-article:7542548 | pubmed:author | pubmed-author:KollerBB | lld:pubmed |
| pubmed-article:7542548 | pubmed:author | pubmed-author:TedderT FTF | lld:pubmed |
| pubmed-article:7542548 | pubmed:author | pubmed-author:ZhouL JLJ | lld:pubmed |
| pubmed-article:7542548 | pubmed:author | pubmed-author:OrdD CDC | lld:pubmed |
| pubmed-article:7542548 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7542548 | pubmed:volume | 3 | lld:pubmed |
| pubmed-article:7542548 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7542548 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7542548 | pubmed:pagination | 39-50 | lld:pubmed |
| pubmed-article:7542548 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:7542548 | pubmed:meshHeading | pubmed-meshheading:7542548-... | lld:pubmed |
| pubmed-article:7542548 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7542548 | pubmed:articleTitle | Abnormal B lymphocyte development, activation, and differentiation in mice that lack or overexpress the CD19 signal transduction molecule. | lld:pubmed |
| pubmed-article:7542548 | pubmed:affiliation | Department of Immunology, Duke University Medical Center, Durham, North Carolina 27710, USA. | lld:pubmed |
| pubmed-article:7542548 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7542548 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:7542548 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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