| pubmed-article:6968813 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:6968813 | lifeskim:mentions | umls-concept:C0039195 | lld:lifeskim |
| pubmed-article:6968813 | lifeskim:mentions | umls-concept:C1509144 | lld:lifeskim |
| pubmed-article:6968813 | lifeskim:mentions | umls-concept:C0205214 | lld:lifeskim |
| pubmed-article:6968813 | lifeskim:mentions | umls-concept:C1709634 | lld:lifeskim |
| pubmed-article:6968813 | lifeskim:mentions | umls-concept:C0337051 | lld:lifeskim |
| pubmed-article:6968813 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:6968813 | pubmed:dateCreated | 1981-1-26 | lld:pubmed |
| pubmed-article:6968813 | pubmed:abstractText | If the collaborative requirement of Lyt 1 T helper cells is bypassed by the Lyt 1 T cell-derived mediator of T help, termed Il-2, upon antigenic stimulation, PNA+ Lyt 123 thymocytes differentiate into either alloreactive or H-2-restricted PNA- Lyt 23 cytotoxic effector cells. Along the differentiation pathway from Lyt 123 leads to 23 effector cells, cytolytic activity is carried out by T cells that still express the Lyt 123 phenotype. The data establish that Lyt 23 CTL are produced by differentiation from antecedent Lyt 123 cells. | lld:pubmed |
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| pubmed-article:6968813 | pubmed:language | eng | lld:pubmed |
| pubmed-article:6968813 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6968813 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:6968813 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6968813 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:6968813 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:6968813 | pubmed:author | pubmed-author:KleinJJ | lld:pubmed |
| pubmed-article:6968813 | pubmed:author | pubmed-author:WagnerHH | lld:pubmed |
| pubmed-article:6968813 | pubmed:author | pubmed-author:RöllinghoffMM | lld:pubmed |
| pubmed-article:6968813 | pubmed:author | pubmed-author:PfizenmaierKK | lld:pubmed |
| pubmed-article:6968813 | pubmed:author | pubmed-author:HardyDD | lld:pubmed |
| pubmed-article:6968813 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6968813 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:6968813 | pubmed:volume | 152 | lld:pubmed |
| pubmed-article:6968813 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6968813 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6968813 | pubmed:pagination | 1413-8 | lld:pubmed |
| pubmed-article:6968813 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:6968813 | pubmed:year | 1980 | lld:pubmed |
| pubmed-article:6968813 | pubmed:articleTitle | Alloreactive and H-2-restricted Lyt 23 cytotoxic T lymphocytes derive from a common pool of antecedent Lyt 123 precursors. | lld:pubmed |
| pubmed-article:6968813 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6968813 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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