| pubmed-article:6795273 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:6795273 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:6795273 | lifeskim:mentions | umls-concept:C0027571 | lld:lifeskim |
| pubmed-article:6795273 | lifeskim:mentions | umls-concept:C0162388 | lld:lifeskim |
| pubmed-article:6795273 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:6795273 | pubmed:dateCreated | 1982-1-28 | lld:pubmed |
| pubmed-article:6795273 | pubmed:abstractText | During the routine examination of a healthy 31-yr-old woman, we found an incomplete deficiency of the 9th component of complement (C9). By hemolytic assay her serum C9 activity was 10 to 15% of normal. Limited family studies suggested that she inherited the deficiency as an autosomal codominant trait. She had no history of unusual or severe infections. When tested for bactericidal activity against serum-sensitive Neisseria gonorrhoeae and N. meningitidis, her serum reacted comparably to normal serum. Normal serum depleted immunochemically of C9 and sera from congenitally C9-deficient patients were also bactericidal against serum-sensitive Neisseria but required 120 min to kill the same numbers of gonococci that intact serum killed within 30 min. In the electron microscope, N. gonorrhoeae incubated with C9-depleted serum were fragmented but lacked the typical C lesions. Therefore, serum lacking C9 can kill serum-sensitive Neisseria, unlike sera deficient in the other terminal C components. | lld:pubmed |
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| pubmed-article:6795273 | pubmed:language | eng | lld:pubmed |
| pubmed-article:6795273 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6795273 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:6795273 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:6795273 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:6795273 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6795273 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:6795273 | pubmed:issn | 0022-1767 | lld:pubmed |
| pubmed-article:6795273 | pubmed:author | pubmed-author:BraudeA IAI | lld:pubmed |
| pubmed-article:6795273 | pubmed:author | pubmed-author:CurdJ GJG | lld:pubmed |
| pubmed-article:6795273 | pubmed:author | pubmed-author:LintT FTF | lld:pubmed |
| pubmed-article:6795273 | pubmed:author | pubmed-author:EsserA FAF | lld:pubmed |
| pubmed-article:6795273 | pubmed:author | pubmed-author:WunderlichA... | lld:pubmed |
| pubmed-article:6795273 | pubmed:author | pubmed-author:PodackE RER | lld:pubmed |
| pubmed-article:6795273 | pubmed:author | pubmed-author:HarrimanG RGR | lld:pubmed |
| pubmed-article:6795273 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6795273 | pubmed:volume | 127 | lld:pubmed |
| pubmed-article:6795273 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6795273 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6795273 | pubmed:pagination | 2386-90 | lld:pubmed |
| pubmed-article:6795273 | pubmed:dateRevised | 2009-11-3 | lld:pubmed |
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| pubmed-article:6795273 | pubmed:meshHeading | pubmed-meshheading:6795273-... | lld:pubmed |
| pubmed-article:6795273 | pubmed:year | 1981 | lld:pubmed |
| pubmed-article:6795273 | pubmed:articleTitle | The role of C9 in complement-mediated killing of Neisseria. | lld:pubmed |
| pubmed-article:6795273 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6795273 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:6795273 | pubmed:publicationType | Case Reports | lld:pubmed |
| pubmed-article:6795273 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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