| pubmed-article:6788878 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:6788878 | lifeskim:mentions | umls-concept:C0007600 | lld:lifeskim |
| pubmed-article:6788878 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:6788878 | lifeskim:mentions | umls-concept:C1536403 | lld:lifeskim |
| pubmed-article:6788878 | lifeskim:mentions | umls-concept:C1522642 | lld:lifeskim |
| pubmed-article:6788878 | lifeskim:mentions | umls-concept:C0034788 | lld:lifeskim |
| pubmed-article:6788878 | lifeskim:mentions | umls-concept:C0205245 | lld:lifeskim |
| pubmed-article:6788878 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
| pubmed-article:6788878 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:6788878 | pubmed:dateCreated | 1981-9-15 | lld:pubmed |
| pubmed-article:6788878 | pubmed:abstractText | To investigate the role of Lyt-2 and Thy-1 in cytolysis, we have generated, by ethyl methanesulfonate mutagenesis and selection, variants of the cloned cytolytic T lymphocyte line L3 that specifically lack either Lyt-2 or Thy-1. An analysis of these variants indicates that neither Lyt-2 nor Lyt-3 is responsible for the lethal hit, but suggests that Lyt-2 and/or Lyt-3 are required for an antigen receptor functional in cytolysis. The data also suggest that the expression of Lyt-3 on the cell surface is not independent of the expression of Lyt-2. Finally the data indicate the Thy-1 plays no role in cytolysis. | lld:pubmed |
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| pubmed-article:6788878 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:6788878 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:6788878 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:6788878 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:6788878 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:6788878 | pubmed:author | pubmed-author:FitchF WFW | lld:pubmed |
| pubmed-article:6788878 | pubmed:author | pubmed-author:MONEYNN | lld:pubmed |
| pubmed-article:6788878 | pubmed:author | pubmed-author:GlasebrookA... | lld:pubmed |
| pubmed-article:6788878 | pubmed:author | pubmed-author:DialynasD PDP | lld:pubmed |
| pubmed-article:6788878 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:6788878 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:6788878 | pubmed:volume | 153 | lld:pubmed |
| pubmed-article:6788878 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:6788878 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:6788878 | pubmed:pagination | 595-604 | lld:pubmed |
| pubmed-article:6788878 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:6788878 | pubmed:year | 1981 | lld:pubmed |
| pubmed-article:6788878 | pubmed:articleTitle | Lyt-2-/Lyt 3- variants of a cloned cytolytic T cell line lack an antigen receptor functional in cytolysis. | lld:pubmed |
| pubmed-article:6788878 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:6788878 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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